Spatiotemporal pattern of neuroinflammation after impact-acceleration closed head injury in the rat. - Wikidata - wikimedia - TriplyDB

Spatiotemporal pattern of neuroinflammation after impact-acceleration closed head injury in the rat.

Spatiotemporal pattern of neuroinflammation after impact-acceleration closed head injury in the rat.

http://www.wikidata.org/entity/Q41900337

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Merging pathology with biomechanics using CHIMERA (Closed-Head Impact Model of Engineered Rotational Acceleration): a novel, surgery-free model of traumatic brain injury.Comparison of humoral neuroinflammation and adhesion molecule expression in two models of experimental intracerebral hemorrhage Increased levels of HMGB1 and pro-inflammatory cytokines in children with febrile seizures.A time course of NADPH-oxidase up-regulation and endothelial nitric oxide synthase activation in the hippocampus following neurotrauma.Tumor necrosis factor alpha and Fas receptor contribute to cognitive deficits independent of cell death after concussive traumatic brain injury in mice.Hypobaric hypoxia exacerbates the neuroinflammatory response to traumatic brain injury.Adenosine kinase facilitated astrogliosis-induced cortical neuronal death in traumatic brain injury.Diffuse traumatic axonal injury in mice induces complex behavioural alterations that are normalized by neutralization of interleukin-1β.

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Spatiotemporal pattern of neuroinflammation after impact-acceleration closed head injury in the rat.

http://www.wikidata.org/entity/Q41900337

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2006 nî lūn-bûn

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2006年の論文

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Spatiotemporal pattern of neur ...... closed head injury in the rat.

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Mediators of Inflammation

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Spatiotemporal pattern of neur ...... closed head injury in the rat.

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Guido Stoll

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Jan Verlooy

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Jos Van Reempts

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Marcel Borgers

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Philippe G Jorens

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Sebastian Jander

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Servan Rooker

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Clinical trials in head injury

Impaired autoregulation of cerebral blood flow in an experimental model of traumatic brain injury.

Traumatic injury induces interleukin-6 production by human astrocytes.

Inflammatory mediators and stroke: new opportunities for novel therapeutics.

Experimental brain injury induces differential expression of tumor necrosis factor-alpha mRNA in the CNS.

Cytokines: coordinators of immune and inflammatory responses.

Cytokines in inflammatory brain lesions: helpful and harmful.

Guidelines for the management of severe head injury. Brain Trauma Foundation.

Increase in IL-6, IL-1 and TNF levels in rat brain following traumatic lesion. Influence of pre- and post-traumatic treatment with Ro5 4864, a peripheral-type (p site) benzodiazepine ligand.

Cortical spreading depression induces proinflammatory cytokine gene expression in the rat brain.

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