Valproate and amitriptyline exert common and divergent influences on global and gene promoter-specific chromatin modifications in rat primary astrocytes. - Wikidata - wikimedia - TriplyDB

Valproate and amitriptyline exert common and divergent influences on global and gene promoter-specific chromatin modifications in rat primary astrocytes.

Valproate and amitriptyline exert common and divergent influences on global and gene promoter-specific chromatin modifications in rat primary astrocytes.

http://www.wikidata.org/entity/Q41903220

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Epigenetics of early child development Epigenetic Modifications of Major Depressive Disorder Current status and future prospects for epigenetic psychopharmacology High-affinity glutamate transporters in chronic pain: an emerging therapeutic target Association of FKBP51 with priming of autophagy pathways and mediation of antidepressant treatment response: evidence in cells, mice, and humans Antidepressants inhibit DNA methyltransferase 1 through reducing G9a levels Histone deacetylase inhibition alters histone methylation associated with heat shock protein 70 promoter modifications in astrocytes and neurons DNA methylation profiling of the fibrinogen gene landscape in human cells and during mouse and zebrafish development.Potential molecular and cellular mechanism of psychotropic drugs Implications and limitations of cellular reprogramming for psychiatric drug development.Epigenetic alterations in depression and antidepressant treatment Amitriptyline up-regulates connexin43-gap junction in rat cultured cortical astrocytes via activation of the p38 and c-Fos/AP-1 signalling pathway Up-regulation of spinal glutamate transporters contributes to anti-hypersensitive effects of valproate in rats after peripheral nerve injury Horizons of psychiatric genetics and epigenetics: where are we and where are we heading?The role of miRNAs in complex formation and control.Intranasally administered neuropeptide S (NPS) exerts anxiolytic effects following internalization into NPS receptor-expressing neurons.Prenatal antiepileptic exposure associates with neonatal DNA methylation differences.DNA Methylation Changes in Valproic Acid-Treated HeLa Cells as Assessed by Image Analysis, Immunofluorescence and Vibrational Microspectroscopy Targeting glia cells: novel perspectives for the treatment of neuropsychiatric diseases.Comparative network-based recovery analysis and proteomic profiling of neurological changes in valproic acid-treated mice.Valproate prevents dysregulation of spinal glutamate and reduces the development of hypersensitivity in rats after peripheral nerve injury.SLC1 glutamate transporters Reactive astrocytes as therapeutic targets for CNS disorders.Epigenetic programming of the HPA axis: early life decides.Sex differences in brain epigenetics.Valproic acid for the treatment of malignant gliomas: review of the preclinical rationale and published clinical results.Lost in translation. New unexplored avenues for neuropsychopharmacology: epigenetics and microRNAs.Epigenetics primer: why the clinician should care about epigenetics.Genetic dys-regulation of astrocytic glutamate transporter EAAT2 and its implications in neurological disorders and manganese toxicity Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease.Valproate increases dopamine transporter expression through histone acetylation and enhanced promoter binding of Nurr1.Glutamate Transport: A New Bench to Bedside Mechanism for Treating Drug Abuse Drugging the pain epigenome.Caregiver maltreatment causes altered neuronal DNA methylation in female rodents.The role of DNA methylation in the pathophysiology and treatment of bipolar disorder.FKBP51 inhibits GSK3β and augments the effects of distinct psychotropic medications.Epigenetic Mechanisms in Psychiatric Diseases and Epigenetic Therapy.Antidepressant drugs diversely affect autophagy pathways in astrocytes and neurons--dissociation from cholesterol homeostasis.Glutamate Transport System as a Novel Therapeutic Target in Chronic Pain: Molecular Mechanisms and Pharmacology.Valproic acid stimulates proliferation of glial precursors during cortical gliogenesis in developing rat.

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P2860

Valproate and amitriptyline exert common and divergent influences on global and gene promoter-specific chromatin modifications in rat primary astrocytes.

http://www.wikidata.org/entity/Q41903220

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Promoter-wide hypermethylation of the ribosomal RNA gene promoter in the suicide brain

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Survival motor neuron gene 2 silencing by DNA methylation correlates with spinal muscular atrophy disease severity and can be bypassed by histone deacetylase inhibition

The reelin and GAD67 promoters are activated by epigenetic drugs that facilitate the disruption of local repressor complexes

The DNA methyltransferases associate with HP1 and the SUV39H1 histone methyltransferase

Histone hyperacetylation induces demethylation of reelin and 67-kDa glutamic acid decarboxylase promoters

Epigenetic programming by maternal behavior

Localized domains of G9a-mediated histone methylation are required for silencing of neuronal genes

Valproic acid defines a novel class of HDAC inhibitors inducing differentiation of transformed cells

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