Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum. - Wikidata - wikimedia - TriplyDB

Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum.

Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum.

http://www.wikidata.org/entity/Q41903981

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Human malarial disease: a consequence of inflammatory cytokine release Thrombopoietin as biomarker and mediator of cardiovascular damage in critical diseases Novel targets for interleukin 18 binding protein Marked elevations in N-terminal brain natriuretic peptide levels in septic shock Clinical review: Myocardial depression in sepsis and septic shock Sepsis-induced myocardial dysfunction: pathophysiology and management Cardiac Troponins in Dogs and Cats A review of sepsis-induced cardiomyopathy Myocardial Dysfunction and Shock after Cardiac Arrest Targeting interleukin-1 in heart disease Overexpression of interleukin-18 aggravates cardiac fibrosis and diastolic dysfunction in fructose-fed rats Lipopolysaccharide improves cardiomyocyte survival and function after serum deprivation Elevated cardiac troponin I in sepsis and septic shock: no evidence for thrombus associated myocardial necrosis Septic cardiomyopathy - A not yet discovered cardiomyopathy?Altered interleukin-1 receptor antagonist and interleukin-18 mRNA expression in myocardial tissues of patients with dilatated cardiomyopathy.Characterization of human septic sera induced gene expression modulation in human myocytes.NOD2-mediated suppression of CD55 on neutrophils enhances C5a generation during polymicrobial sepsis.Can Vaccinations Improve Heart Failure Outcomes?: Contemporary Data and Future Directions.Caspase inhibition prevents cardiac dysfunction and heart apoptosis in a rat model of sepsis.The association between pneumococcal pneumonia and acute cardiac events.Myocardial dysfunction in sepsis: a large, unsolved puzzle Cardiac Troponin Release is Associated with Biomarkers of Inflammation and Ventricular Dilatation During Critical Illness.Deeper understanding of mechanisms contributing to sepsis-induced myocardial dysfunction.Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes Update on meningococcal disease with emphasis on pathogenesis and clinical management Roles of tumor necrosis factor alpha (TNF-alpha) and the p55 TNF receptor in CD1d induction and coxsackievirus B3-induced myocarditis.Management of the critically ill patient with severe sepsis.Postinfarct intramyocardial injection of mesenchymal stem cells pretreated with TGF-alpha improves acute myocardial function.Aging and animal models of systemic insult: trauma, burn, and sepsis.Bench to bedside: tumor necrosis factor-alpha: from inflammation to resuscitation.Hypoxia-inducible factor 2alpha regulates macrophage function in mouse models of acute and tumor inflammation Effect of glucose-insulin-potassium infusion on mortality in critical care settings: a systematic review and meta-analysis.Interleukin-1β reduces L-type Ca2+ current through protein kinase Cϵ activation in mouse heart.Cardiac function in Vietnamese patients with different dengue severity grades.Severe postpartum sepsis with prolonged myocardial dysfunction: a case report.Enhanced monocyte chemoattractant protein-1 production in aging mice exaggerates cardiac depression during endotoxemia TLR2 stimulation induces cardiac inflammation but not cardiac depression in vivo.Inhibition of caspase 1 reduces human myocardial ischemic dysfunction via inhibition of IL-18 and IL-1beta The role of statins in prevention and treatment of community acquired pneumonia: a systematic review and meta-analysis.Modulation of the triggering receptor expressed on the myeloid cell type 1 pathway in murine septic shock

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P2860

Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum.

http://www.wikidata.org/entity/Q41903981

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1996 nî lūn-bûn

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Tumor necrosis factor alpha an ...... d by human septic shock serum.

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Dee L

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Kumar A

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Olson J

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Parrillo JE

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Uretz E

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P2860

Interleukin 1 induces a shock-like state in rabbits. Synergism with tumor necrosis factor and the effect of cyclooxygenase inhibition

Abnormal contractile function due to induction of nitric oxide synthesis in rat cardiac myocytes follows exposure to activated macrophage-conditioned medium.

Cellular basis for the negative inotropic effects of tumor necrosis factor-alpha in the adult mammalian heart.

Interleukin 1 and tumor necrosis factor inhibit cardiac myocyte beta-adrenergic responsiveness

A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance

Gram-negative bacteremia produces both severe systolic and diastolic cardiac dysfunction in a canine model that simulates human septic shock

The complex pattern of cytokines in serum from patients with meningococcal septic shock. Association between interleukin 6, interleukin 1, and fatal outcome.

Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock.

Effects of cytokines on the contractility of cultured cardiac myocytes.

Immediate release of a nitric oxide-like factor from bovine aortic endothelial cells by Escherichia coli lipopolysaccharide.

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