The transcription factor cyclic AMP-responsive element-binding protein H regulates triglyceride metabolism.
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Integrated physiology and systems biology of PPARαResearch Perspectives on the Regulation and Physiological Functions of FGF21 and its Association with NAFLDEnergy metabolism in the liverTranscriptional regulation of fibroblast growth factor 21 expressionCREBH determines the severity of sulpyrine-induced fatal shockProatherogenic abnormalities of lipid metabolism in SirT1 transgenic mice are mediated through Creb deacetylationThe Role and Potential Therapeutic Implications of the Fibroblast Growth Factors in Energy Balance and Type 2 Diabetes.Endoplasmic reticulum stress and oxidative stress in cell fate decision and human disease.The polygenic nature of hypertriglyceridaemia: implications for definition, diagnosis, and management.Circadian regulators of intestinal lipid absorption.Tissue-specific actions of the metabolic hormones FGF15/19 and FGF21.Hepatic cannabinoid receptor type 1 mediates alcohol-induced regulation of bile acid enzyme genes expression via CREBH.GCN2 in the brain programs PPARγ2 and triglyceride storage in the liver during perinatal development in response to maternal dietary fat.Transcriptional activation of Fsp27 by the liver-enriched transcription factor CREBH promotes lipid droplet growth and hepatic steatosis.A liver-enriched long non-coding RNA, lncLSTR, regulates systemic lipid metabolism in mice.Botanical oils enriched in n-6 and n-3 FADS2 products are equally effective in preventing atherosclerosis and fatty liverExcess of rare variants in non-genome-wide association study candidate genes in patients with hypertriglyceridemiaEndoplasmic reticulum-tethered transcription factor cAMP responsive element-binding protein, hepatocyte specific, regulates hepatic lipogenesis, fatty acid oxidation, and lipolysis upon metabolic stress in mice.Fasting-induced G0/G1 switch gene 2 and FGF21 expression in the liver are under regulation of adipose tissue derived fatty acids.Parasite-induced ER stress response in hepatocytes facilitates Plasmodium liver stage infection.The role of CREB-H transcription factor in triglyceride metabolism.New wrinkles in lipoprotein lipase biology.The impact of the unfolded protein response on human diseasePhosphorylation and SCF-mediated degradation regulate CREB-H transcription of metabolic targets.Lysine Acetylation of CREBH Regulates Fasting-Induced Hepatic Lipid Metabolism.Activation of cannabinoid receptor type 1 (Cb1r) disrupts hepatic insulin receptor signaling via cyclic AMP-response element-binding protein H (Crebh)-mediated induction of Lipin1 gene.IRE1α-XBP1s induces PDI expression to increase MTP activity for hepatic VLDL assembly and lipid homeostasis.An orchestrated program regulating secretory pathway genes and cargos by the transmembrane transcription factor CREB-H.β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.Hyperlipidemia and hepatitis in liver-specific CREB3L3 knockout mice generated using a one-step CRISPR/Cas9 systemCREBH-FGF21 axis improves hepatic steatosis by suppressing adipose tissue lipolysisIdentification and characterization of cyclic AMP response element-binding protein H response element in the human apolipoprotein A5 gene promoter.Intestinal CREBH overexpression prevents high-cholesterol diet-induced hypercholesterolemia by reducing Npc1l1 expression.Unfolded protein response signaling and metabolic diseases.Orphan nuclear receptor Errγ induces C-reactive protein gene expression through induction of ER-bound Bzip transmembrane transcription factor CREBH.Liver-enriched transcription factor CREBH interacts with peroxisome proliferator-activated receptor α to regulate metabolic hormone FGF21.Activation of intestinal peroxisome proliferator-activated receptor-α increases high-density lipoprotein production.Transcriptional regulation of apolipoprotein A-IV by the transcription factor CREBHThe complex genetic basis of plasma triglycerides.Targeting endoplasmic reticulum stress in metabolic disease.
P2860
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P2860
The transcription factor cyclic AMP-responsive element-binding protein H regulates triglyceride metabolism.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
The transcription factor cycli ...... lates triglyceride metabolism.
@en
type
label
The transcription factor cycli ...... lates triglyceride metabolism.
@en
prefLabel
The transcription factor cycli ...... lates triglyceride metabolism.
@en
P2093
P2860
P356
P1433
P1476
The transcription factor cycli ...... lates triglyceride metabolism.
@en
P2093
Ann-Hwee Lee
Christopher T Johansen
Jonathan D Brown
Jorge Plutzky
Jung Hoon Lee
Laurie H Glimcher
Petros Giannikopoulos
P2860
P2888
P304
P356
10.1038/NM.2347
P407
P577
2011-06-12T00:00:00Z