Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin. - Wikidata - wikimedia - TriplyDB

Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

http://www.wikidata.org/entity/Q41912941

about

The evolution of vertebrate Toll-like receptors Plasmodium falciparum histones induce endothelial proinflammatory response and barrier dysfunction Pathogen recognition and inflammatory signaling in innate immune defenses Expression and distribution of Toll-like receptors 11-13 in the brain during murine neurocysticercosis Hemozoin: oil versus water Molecular mechanisms of autoimmunity triggered by microbial infection.Dendritic Cells and Their Multiple Roles during Malaria Infection Genetic polymorphisms linked to susceptibility to malaria Role of interleukin-10 in malaria: focusing on coinfection with lethal and nonlethal murine malaria parasites Both functional LTbeta receptor and TNF receptor 2 are required for the development of experimental cerebral malaria Splenic red pulp macrophages produce type I interferons as early sentinels of malaria infection but are dispensable for control Insufficiently defined genetic background confounds phenotypes in transgenic studies as exemplified by malaria infection in Tlr9 knockout mice A Novel Toll-Like Receptor (TLR) Influences Compatibility between the Gastropod Biomphalaria glabrata, and the Digenean Trematode Schistosoma mansoni Malaria impairs T cell clustering and immune priming despite normal signal 1 from dendritic cells Malaria parasite tyrosyl-tRNA synthetase secretion triggers pro-inflammatory responses Plasmodium falciparum merozoite surface protein 1 blocks the proinflammatory protein S100P Toll-like receptor (TLR) polymorphisms in African children: Common TLR-4 variants predispose to severe malaria.TLR signaling Phosphoinositide 3-kinase-dependent inhibition of dendritic cell interleukin-12 production by Giardia lamblia Regulation of immunopathogenesis during Plasmodium and Toxoplasma infections: more parallels than distinctions?Malarial hemozoin: from target to tool Splenic differentiation and emergence of CCR5+CXCL9+CXCL10+ monocyte-derived dendritic cells in the brain during cerebral malaria.Cross-talk with myeloid accessory cells regulates human natural killer cell interferon-gamma responses to malaria.Plasmodium strain determines dendritic cell function essential for survival from malaria Plasmodium-induced inflammation by uric acid Murine cerebral malaria development is independent of toll-like receptor signaling Blood coagulation, inflammation, and malaria.Pathogenesis of malaria in tissues and blood.Alterations in cytokines and haematological parameters during the acute and convalescent phases of Plasmodium falciparum and Plasmodium vivax infections.Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.Malarial hemozoin is a Nalp3 inflammasome activating danger signal.Synthetic Plasmodium-like hemozoin activates the immune response: a morphology - function study Gastrointestinal nematode infection exacerbates malaria-induced liver pathology.Plasmepsin 4-deficient Plasmodium berghei are virulence attenuated and induce protective immunity against experimental malaria.Elevated levels of vascular endothelial growth factor (VEGF) and soluble vascular endothelial growth factor receptor (VEGFR)-2 in human malaria.A randomized and controlled Phase 1 study of the safety and immunogenicity of the AMA1-C1/Alhydrogel + CPG 7909 vaccine for Plasmodium falciparum malaria in semi-immune Malian adults.Parasite-derived plasma microparticles contribute significantly to malaria infection-induced inflammation through potent macrophage stimulation The role of lipopeptidophosphoglycan in the immune response to Entamoeba histolytica.Toll-Interacting Protein in Resolving and Non-Resolving Inflammation.A surprising role for uric acid: the inflammatory malaria response.

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P2860

Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

http://www.wikidata.org/entity/Q41912941

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Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

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label

Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

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Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

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Journal of Experimental Medicine

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Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin.

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Hiroaki Hemmi

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Masahiro Yamamoto

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Osamu Takeuchi

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Satoshi Uematsu

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Sawako Itagaki

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Shintaro Sato

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Taro Kawai

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P2860

A Toll-like receptor recognizes bacterial DNA

Toll-like receptor signalling

On the molecular mechanism of chloroquine's antimalarial action

Innate immune recognition

Small anti-viral compounds activate immune cells via the TLR7 MyD88-dependent signaling pathway

Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components

Role of adaptor TRIF in the MyD88-independent toll-like receptor signaling pathway

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

Purified malaria pigment (hemozoin) enhances dendritic cell maturation and modulates the isotype of antibodies induced by a DNA vaccine

Theories on malarial pigment formation and quinoline action.

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19-25

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10.1084/JEM.20041836

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1

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201

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Toshihiro Horii

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8101134

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15630134

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P921

Toll-like receptor

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2212757

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