IFN-α-mediated Base Excision Repair Pathway Correlates with Antiviral Response Against Hepatitis B Virus Infection.

about

IFN-α-mediated Base Excision Repair Pathway Correlates with Antiviral Response Against Hepatitis B Virus Infection.

Item

http://www.wikidata.org/entity/Q41923245

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

IFN-α-mediated Base Excision R ...... t Hepatitis B Virus Infection.

@en

type

Item

label

IFN-α-mediated Base Excision R ...... t Hepatitis B Virus Infection.

@en

prefLabel

IFN-α-mediated Base Excision R ...... t Hepatitis B Virus Infection.

@en

P1476

IFN-α-mediated Base Excision R ...... st Hepatitis B Virus Infection

@en

P2093

Guang Chen

http://www.w3.org/2001/XMLSchema#string

Qin Ning

http://www.w3.org/2001/XMLSchema#string

Wolfgang E Thasler

http://www.w3.org/2001/XMLSchema#string

Yong Li

http://www.w3.org/2001/XMLSchema#string

P2860

MBD4 and TDG: multifaceted DNA glycosylases with ever expanding biological roles

InSilico DB genomic datasets hub: an efficient starting point for analyzing genome-wide studies in GenePattern, Integrative Genomics Viewer, and R/Bioconductor.

Differential interferon signaling in liver lobule and portal area cells under treatment for chronic hepatitis C.

Interferons: Success in anti-viral immunotherapy.

Neil3 and NEIL1 DNA glycosylases remove oxidative damages from quadruplex DNA and exhibit preferences for lesions in the telomeric sequence context.

The hepatitis B virus x protein inhibits thymine DNA glycosylase initiated base excision repair

Uracil DNA glycosylase counteracts APOBEC3G-induced hypermutation of hepatitis B viral genomes: excision repair of covalently closed circular DNA.

Hypermutation of hepatitis B virus genomes by APOBEC3G, APOBEC3C and APOBEC3H.

Control of Hepatitis B Virus by Cytokines

Secreted Interferon-Inducible Factors Restrict Hepatitis B and C Virus Entry In Vitro.

P2888

s41598-017-13082-z

P304

12715

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/S41598-017-13082-Z

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

P577

2017-10-05T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28983111

http://www.w3.org/2001/XMLSchema#string

P932

5629255

http://www.w3.org/2001/XMLSchema#string