Pneumolysin causes neuronal cell death through mitochondrial damage. - Wikidata - wikimedia - TriplyDB

Pneumolysin causes neuronal cell death through mitochondrial damage.

Pneumolysin causes neuronal cell death through mitochondrial damage.

http://www.wikidata.org/entity/Q41960118

about

Pathophysiology and treatment of bacterial meningitis More than a pore: the cellular response to cholesterol-dependent cytolysins The MACPF/CDC family of pore-forming toxins.Pneumolysin activates the NLRP3 inflammasome and promotes proinflammatory cytokines independently of TLR4.Thrombopoietin contributes to neuronal damage in experimental bacterial meningitis.Bacterial pore-forming cytolysins induce neuronal damage in a rat model of neonatal meningitis.Two DHH subfamily 1 proteins contribute to pneumococcal virulence and confer protection against pneumococcal disease.Pathogenesis of A Clinical Ocular Strain of Streptococcus pneumoniae and the Interaction of Pneumolysin with Corneal Cells.Household air pollution causes dose-dependent inflammation and altered phagocytosis in human macrophages.Pore-Forming Toxins Induce Macrophage Necroptosis during Acute Bacterial Pneumonia.Review: apoptotic mechanisms in bacterial infections of the central nervous system.Pneumococcal Pneumolysin Induces DNA Damage and Cell Cycle Arrest The influence of natural lipid asymmetry upon the conformation of a membrane-inserted protein (perfringolysin O)Targeting of Helicobacter pylori VacA to mitochondria.Bacterial toxins and the nervous system: neurotoxins and multipotential toxins interacting with neuronal cells.Chronicle of a death foretold: Plasmodium liver stage parasites decide on the fate of the host cell.Hijacking mitochondria: bacterial toxins that modulate mitochondrial function.Listeria infection modulates mitochondrial dynamics.A novel schiff base zinc coordination compound inhibits proliferation and induces apoptosis of human osteosarcoma cells.Could proteomic research deliver the next generation of treatments for pneumococcal meningitis?Effect of Escherichia coli STb toxin on NIH-3T3 cells.Pneumolysin activates macrophage lysosomal membrane permeabilization and executes apoptosis by distinct mechanisms without membrane pore formation.Export requirements of pneumolysin in Streptococcus pneumoniae.Pore-forming toxin-mediated ion dysregulation leads to death receptor-independent necroptosis of lung epithelial cells during bacterial pneumonia.Joint analysis of SNPs and proteins identifies regulatory IL18 gene variations decreasing the chance of spastic cerebral palsy.Radical scavenging, prolyl endopeptidase inhibitory, and antimicrobial potential of a cultured Himalayan lichen Cetrelia olivetorum.Pneumolysin induced mitochondrial dysfunction leads to release of mitochondrial DNA.Inducible nitric oxide synthase mediates hippocampal caspase-3 activation in pneumococcal meningitis.Hsp70 Suppresses Mitochondrial Reactive Oxygen Species and Preserves Pulmonary Microvascular Barrier Integrity Following Exposure to Bacterial Toxins.

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P2860

Pneumolysin causes neuronal cell death through mitochondrial damage.

http://www.wikidata.org/entity/Q41960118

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Pneumolysin causes neuronal cell death through mitochondrial damage.

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Pneumolysin causes neuronal cell death through mitochondrial damage.

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Pneumolysin causes neuronal cell death through mitochondrial damage.

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Infection and Immunity

P1476

Pneumolysin causes neuronal cell death through mitochondrial damage.

@en

P2093

Daniela Bermpohl

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Dorette Freyer

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Emilie Dagand

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Ingo Bechmann

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Joerg R Weber

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Johann S Braun

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Miriam Schickhaus

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Olaf Hoffmann

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P2860

Molecular characterization of mitochondrial apoptosis-inducing factor

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

Two distinct pathways leading to nuclear apoptosis

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

Separation of yeast chromosome-sized DNAs by pulsed field gradient gel electrophoresis

Pneumococcal pneumolysin and H(2)O(2) mediate brain cell apoptosis during meningitis

The mitochondrial permeability transition pore and its role in cell death

Proinflammatory interactions of pneumolysin with human neutrophils.

Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells.

The apoptotic response to pneumolysin is Toll-like receptor 4 dependent and protects against pneumococcal disease

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4245-4254

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scholarly article

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10.1128/IAI.00031-07

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9

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75

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Timothy J Mitchell

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2007-06-11T00:00:00Z

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17562768

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1951198

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