Overexpression of endothelial nitric oxide synthase in endothelial cells is protective against ischemia-reperfusion injury in mouse skeletal muscle.
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Endothelial Barrier and Its Abnormalities in Cardiovascular DiseaseRole of blood cells in ischaemia-reperfusion induced endothelial barrier failure.Expression of key regulators of mitochondrial biogenesis in growth hormone receptor knockout (GHRKO) mice is enhanced but is not further improved by other potential life-extending interventions.Emerging role of PKA/eNOS pathway in therapeutic angiogenesis for ischaemic tissue diseasesHypercholesterolemia: its impact on ischemia-reperfusion injury.Activation of Hsp90-eNOS and increased NO generation attenuate respiration of hypoxia-treated endothelial cellsEndothelial-specific expression of mitochondrial thioredoxin promotes ischemia-mediated arteriogenesis and angiogenesis.Gender-specific modulation of the response to arterial injury by soluble guanylate cyclase α1.Endothelial dysfunction as a cellular mechanism for vascular failure.Mechanisms of muscle injury, repair, and regeneration.The Gastrointestinal Circulation: Physiology and Pathophysiology.Critical O2 and NO concentrations in NO-induced cell death in a rat liver sinusoidal endothelial cell line.Nitric oxide is a mediator of antiproliferative effects induced by proinflammatory cytokines on pancreatic beta cells.Nitric Oxide is Necessary for Diazoxide Protection Against Ischemic Injury in Skeletal Muscle.Endothelial nitric oxide synthase overexpression attenuates myocardial reperfusion injury.Inhibition of iNOS with 1400W improves contractile function and alters nos gene and protein expression in reperfused skeletal muscle.Control of myocardial oxygen consumption in transgenic mice overexpressing vascular eNOS.Addition of nitric oxide donor S-nitroso-N-acetylcysteine to selective iNOS inhibitor 1400W further improves contractile function in reperfused skeletal muscle.Pravastatin attenuates tourniquet-induced skeletal muscle ischemia reperfusion injury.Production of endothelial nitric oxide synthase (eNOS) over-expressing piglets.Effects of non contact low-frequency ultrasound on healing of suspected deep tissue injury: a retrospective analysis.Nitrite as a pharmacological intervention for the successful treatment of crush syndrome.Limb Ischemic Conditioning Improved Cognitive Deficits via eNOS-Dependent Augmentation of Angiogenesis after Chronic Cerebral Hypoperfusion in RatsThe Protective Effect of the Total Flavonoids of L. Flowers on Transient Cerebral Ischemia-Reperfusion Injury Is due to Activation of the Nrf2-ARE Pathway
P2860
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P2860
Overexpression of endothelial nitric oxide synthase in endothelial cells is protective against ischemia-reperfusion injury in mouse skeletal muscle.
description
2002 nî lūn-bûn
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2002年の論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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2002年论文
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2002年论文
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2002年论文
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name
Overexpression of endothelial ...... jury in mouse skeletal muscle.
@en
type
label
Overexpression of endothelial ...... jury in mouse skeletal muscle.
@en
prefLabel
Overexpression of endothelial ...... jury in mouse skeletal muscle.
@en
P2093
P2860
P921
P1476
Overexpression of endothelial ...... jury in mouse skeletal muscle.
@en
P2093
Ken-ichi Hirata Ki
Masanori Ozaki
Masayuki Namiki
Mitsuhiro Yokoyama
Nobutaka Inoue
Seinosuke Kawashima
Tetsuaki Hirase
Tomoya Yamashita
P2860
P304
P356
10.1016/S0002-9440(10)62560-6
P407
P577
2002-04-01T00:00:00Z