West Nile virus differentially modulates the unfolded protein response to facilitate replication and immune evasion.
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Stress responses in flavivirus-infected cells: activation of unfolded protein response and autophagyCellular vimentin regulates construction of dengue virus replication complexes through interaction with NS4A proteinApoptosis, autophagy and unfolded protein response pathways in Arbovirus replication and pathogenesisAlphavirus Infection: Host Cell Shut-Off and Inhibition of Antiviral ResponsesFlaviviral NS4b, chameleon and jack-in-the-box roles in viral replication and pathogenesis, and a molecular target for antiviral interventionWest Nile virus adaptation to ixodid tick cells is associated with phenotypic trade-offs in primary hostsHepatitis C Virus Infection Induces Autophagy as a Prosurvival Mechanism to Alleviate Hepatic ER-Stress ResponseInflammatory and oxidative stress in rotavirus infectionEmerging functions of the unfolded protein response in immunityModulation of the unfolded protein response by the human hepatitis B virusThe many faces of the flavivirus NS5 protein in antagonism of type I interferon signalingAntiviral activity of a small molecule deubiquitinase inhibitor occurs via induction of the unfolded protein responseEarly dengue virus protein synthesis induces extensive rearrangement of the endoplasmic reticulum independent of the UPR and SREBP-2 pathwayPorcine circovirus type 2 capsid protein induces unfolded protein response with subsequent activation of apoptosis.Coronavirus infection, ER stress, apoptosis and innate immunityER stress, autophagy, and RNA viruses.West Nile virus replication requires fatty acid synthesis but is independent on phosphatidylinositol-4-phosphate lipidsNovel approaches to flavivirus drug discovery.Activation of ERAD pathway by human hepatitis B virus modulates viral and subviral particle production.The cellular redox environment alters antigen presentationIRF-3, IRF-5, and IRF-7 coordinately regulate the type I IFN response in myeloid dendritic cells downstream of MAVS signalingInfection with Usutu virus induces an autophagic response in mammalian cells.The cell biology of the unfolded protein response.Constant up-regulation of BiP/GRP78 expression prevents virus-induced apoptosis in BHK-21 cells with Japanese encephalitis virus persistent infection.A conserved peptide in West Nile virus NS4A protein contributes to proteolytic processing and is essential for replicationHeterogeneous nuclear ribonucleoprotein A2 participates in the replication of Japanese encephalitis virus through an interaction with viral proteins and RNA.Characterization of dengue virus NS4A and NS4B protein interaction.HIV infection and antiretroviral therapy lead to unfolded protein response activation.The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus InfectionWest Nile virus population genetics and evolution.West nile virus infections suppress early viral RNA synthesis and avoid inducing the cell stress granule response.Flavivirus Antagonism of Type I Interferon Signaling Reveals Prolidase as a Regulator of IFNAR1 Surface Expression.Mouse norovirus 1 utilizes the cytoskeleton network to establish localization of the replication complex proximal to the microtubule organizing centerInduction of the cellular microRNA, Hs_154, by West Nile virus contributes to virus-mediated apoptosis through repression of antiapoptotic factors.Murine cytomegalovirus targets transcription factor ATF4 to exploit the unfolded-protein responseMutational analysis of the West Nile virus NS4B protein.Evidence for a genetic and physical interaction between nonstructural proteins NS1 and NS4B that modulates replication of West Nile virusArsenite-induced stress granule formation is inhibited by elevated levels of reduced glutathione in West Nile virus-infected cells.JNK1 Derived from Orange-Spotted Grouper, Epinephelus coioides, Involving in the Evasion and Infection of Singapore Grouper Iridovirus (SGIV).ATF6 signaling is required for efficient West Nile virus replication by promoting cell survival and inhibition of innate immune responses.
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West Nile virus differentially modulates the unfolded protein response to facilitate replication and immune evasion.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
West Nile virus differentially ...... eplication and immune evasion.
@en
type
label
West Nile virus differentially ...... eplication and immune evasion.
@en
prefLabel
West Nile virus differentially ...... eplication and immune evasion.
@en
P2860
P356
P1433
P1476
West Nile virus differentially ...... eplication and immune evasion.
@en
P2093
Rebecca L Ambrose
P2860
P304
P356
10.1128/JVI.02050-10
P407
P577
2010-12-29T00:00:00Z