Absence of exogenous interleukin-4-induced apoptosis of gingival macrophages may contribute to chronic inflammation in periodontal diseases. - Wikidata - wikimedia - TriplyDB

Absence of exogenous interleukin-4-induced apoptosis of gingival macrophages may contribute to chronic inflammation in periodontal diseases.

Absence of exogenous interleukin-4-induced apoptosis of gingival macrophages may contribute to chronic inflammation in periodontal diseases.

http://www.wikidata.org/entity/Q41973681

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In vitro induction of activation-induced cell death in lymphocytes from chronic periodontal lesions by exogenous Fas ligand Alkaline conditions accelerate polymorphonuclear leukocyte apoptosis in vitro.Targeted salivary gland immunization with plasmid DNA elicits specific salivary immunoglobulin A and G antibodies and serum immunoglobulin G antibodies in mice Serum immunoglobulin G (IgG) and IgG subclass responses to the RgpA-Kgp proteinase-adhesin complex of Porphyromonas gingivalis in adult periodontitis Hyper-responsive phenotype in localized aggressive periodontitis.Combined stimulation with the T helper cell type 2 cytokines interleukin (IL)-4 and IL-10 induces mouse mast cell apoptosis Mutants in the ADP-ribosyltransferase cleft of cholera toxin lack diarrheagenicity but retain adjuvanticity A nontoxic mutant of cholera toxin elicits Th2-type responses for enhanced mucosal immunity.Immuno-pathogenesis of Periodontal Disease: Current and Emerging Paradigms

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P2860

Absence of exogenous interleukin-4-induced apoptosis of gingival macrophages may contribute to chronic inflammation in periodontal diseases.

http://www.wikidata.org/entity/Q41973681

description

1996 nî lūn-bûn

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1996年の論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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1996年論文

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1996年论文

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1996年论文

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1996年论文

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name

Absence of exogenous interleuk ...... ation in periodontal diseases.

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Absence of exogenous interleuk ...... ation in periodontal diseases.

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Absence of exogenous interleuk ...... ation in periodontal diseases.

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The American Journal of Pathology

P1476

Absence of exogenous interleuk ...... ation in periodontal diseases.

@en

P2093

H Kiyono

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J R McGhee

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K Fujihashi

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K Kawabata

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M Yamamoto

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T E Van Dyke

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T Hiroi

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T V Bamberg

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P2860

Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics

Suppression of monocyte function and differential regulation of IL-1 and IL-1ra by IL-4 contribute to resolution of experimental arthritis

Potential antiinflammatory effects of interleukin 4: suppression of human monocyte tumor necrosis factor alpha, interleukin 1, and prostaglandin E2

Regulation of human peripheral blood monocyte DR antigen expression in vitro by lymphokines and recombinant interferons.

Gingival mononuclear cells from chronic inflammatory periodontal tissues produce interleukin (IL)-5 and IL-6 but not IL-2 and IL-4.

Multi-target PCR analysis by capillary electrophoresis and laser-induced fluorescence.

Periodontal disease.

The cell biology of macrophage activation.

Interleukin-1 mRNA-expressing macrophages in human chronically inflamed gingival tissues

Lipopolysaccharide, tumor necrosis factor-alpha, and IL-1 beta prevent programmed cell death (apoptosis) in human peripheral blood monocytes.

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331-339

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scholarly article

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1

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148

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8546223

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1861600

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