Insulin receptor compensates for IGF1R inhibition and directly induces mitogenic activity in prostate cancer cells. - Wikidata - wikimedia - TriplyDB

Insulin receptor compensates for IGF1R inhibition and directly induces mitogenic activity in prostate cancer cells.

Insulin receptor compensates for IGF1R inhibition and directly induces mitogenic activity in prostate cancer cells.

http://www.wikidata.org/entity/Q41985108

about

IGF-IR Targeted Therapy: Past, Present and Future Highly specific role of the insulin receptor in breast cancer progression.Insulin-like growth factor-1 signaling in renal cell carcinoma A novel long noncoding RNA IRAIN regulates cell proliferation in non small cell lung cancer.Disruption of insulin receptor function inhibits proliferation in endocrine-resistant breast cancer cells Space radiation exposure persistently increased leptin and IGF1 in serum and activated leptin-IGF1 signaling axis in mouse intestine Minireview: Were the IGF Signaling Inhibitors All Bad?Snail-Modulated MicroRNA 493 Forms a Negative Feedback Loop with the Insulin-Like Growth Factor 1 Receptor Pathway and Blocks Tumorigenesis MiR-195 inhibits the growth and metastasis of NSCLC cells by targeting IGF1R.Concurrent Diabetes Mellitus may Negatively Influence Clinical Progression and Response to Androgen Deprivation Therapy in Patients with Advanced Prostate Cancer.Investigating the role of the IGF axis as a predictor of biochemical recurrence in prostate cancer patients post-surgery.Oral insulin delivery: existing barriers and current counter-strategies.

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Insulin receptor compensates for IGF1R inhibition and directly induces mitogenic activity in prostate cancer cells.

http://www.wikidata.org/entity/Q41985108

description

2014 nî lūn-bûn

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Endocrine connections

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Doron Weinstein

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Haim Werner

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Rive Sarfstein

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P2860

Insulin signalling and the regulation of glucose and lipid metabolism

Increased insulin-like growth factor I receptor expression and signaling are components of androgen-independent progression in a lineage-derived prostate cancer progression model

Evidence that insulin-like growth factor I and growth hormone are required for prostate gland development

Insulin-like growth factors, their binding proteins, and prostate cancer risk: analysis of individual patient data from 12 prospective studies.

Insulin: new roles for an ancient hormone.

Structure of the human insulin receptor gene and characterization of its promoter.

Androgens up-regulate the insulin-like growth factor-I receptor in prostate cancer cells.

Insulin: discovery and controversy.

Establishment and characterization of a human prostatic carcinoma cell line (PC-3).

Prediagnostic body-mass index, plasma C-peptide concentration, and prostate cancer-specific mortality in men with prostate cancer: a long-term survival analysis.

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24-35

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10.1530/EC-13-0086

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1

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