Add-on aliskiren elicits stronger renoprotection than high-dose valsartan in type 2 diabetic KKAy mice that do not respond to low-dose valsartan.
about
Hyperglycemia causes cellular senescence via a SGLT2- and p21-dependent pathway in proximal tubules in the early stage of diabetic nephropathy.Effects of the Angiotensin receptor blocker olmesartan on adipocyte hypertrophy and function in mice with metabolic disorders.Calcium channel blocker enhances beneficial effects of an angiotensin II AT1 receptor blocker against cerebrovascular-renal injury in type 2 diabetic miceAngiotensin II blockade and renal protection.The diabetes pandemic suggests unmet needs for 'CKD with diabetes' in addition to 'diabetic nephropathy'-implications for pre-clinical research and drug testing.Angiotensin-converting enzyme inhibitor does not suppress renal angiotensin II levels in angiotensin I-infused rats.
P2860
Add-on aliskiren elicits stronger renoprotection than high-dose valsartan in type 2 diabetic KKAy mice that do not respond to low-dose valsartan.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
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name
Add-on aliskiren elicits stron ...... respond to low-dose valsartan.
@en
type
label
Add-on aliskiren elicits stron ...... respond to low-dose valsartan.
@en
prefLabel
Add-on aliskiren elicits stron ...... respond to low-dose valsartan.
@en
P2093
P2860
P356
P1476
Add-on aliskiren elicits stron ...... respond to low-dose valsartan.
@en
P2093
Akira Nishiyama
Daisuke Nakano
Hirofumi Hitomi
Hirohito Mori
Kento Kitada
Tsutomu Masaki
Yu-Yan Fan
P2860
P304
P356
10.1254/JPHS.12031FP
P577
2012-05-22T00:00:00Z