Hypoxia in high glucose followed by reoxygenation in normal glucose reduces the viability of cortical astrocytes through increased permeability of connexin 43 hemichannels.
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Role of Astroglial Hemichannels and Pannexons in Memory and Neurodegenerative DiseasesTherapeutic Hypothermia for Neonatal Hypoxic-Ischemic Encephalopathy - Where to from Here?Intracellular Cleavage of the Cx43 C-Terminal Domain by Matrix-Metalloproteases: A Novel Contributor to Inflammation?Gap junction modulation and its implications for heart functionGap junctionsFrom mechanosensitivity to inflammatory responses: new players in the pathology of glaucomaDisruption in connexin-based communication is associated with intracellular Ca²⁺ signal alterations in astrocytes from Niemann-Pick type C miceThe Role of Connexins in Wound Healing and Repair: Novel Therapeutic ApproachesExtracellular gentamicin reduces the activity of connexin hemichannels and interferes with purinergic Ca(2+) signaling in HeLa cells.Ammonia mediates cortical hemichannel dysfunction in rodent models of chronic liver disease.Connexin hemichannel blockade is neuroprotective after asphyxia in preterm fetal sheepThe role of gap junction channels during physiologic and pathologic conditions of the human central nervous systemHyperglycemia as a Risk Factor of Ischemic StrokePrenatal nicotine exposure enhances Cx43 and Panx1 unopposed channel activity in brain cells of adult offspring mice fed a high-fat/cholesterol diet.ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannelsIntercellular redistribution of cAMP underlies selective suppression of cancer cell growth by connexin26.HIV increases the release of dickkopf-1 protein from human astrocytes by a Cx43 hemichannel-dependent mechanismElevated Intracellular Ca(2+) Signals by Oxidative Stress Activate Connexin 43 Hemichannels in Osteocytes.Role of Akt and Ca2+ on cell permeabilization via connexin43 hemichannels induced by metabolic inhibition.Connexin 43 stabilizes astrocytes in a stroke-like milieu to facilitate neuronal recovery.Deleterious effects of high dose connexin 43 mimetic peptide infusion after cerebral ischaemia in near-term fetal sheep.Connexin and pannexin hemichannels in inflammatory responses of glia and neurons.Connexin and pannexin hemichannels in brain glial cells: properties, pharmacology, and roles.Possible involvement of TLRs and hemichannels in stress-induced CNS dysfunction via mastocytes, and glia activation.The role of the innate immune system in Alzheimer's disease and frontotemporal lobar degeneration: an eye on microglia.Mechanisms of HIV Neuropathogenesis: Role of Cellular Communication Systems.Molecular pathways of pannexin1-mediated neurotoxicity.Connexins and their channels in inflammation.The acute inhibition of enteric glial metabolism with fluoroacetate alters calcium signaling, hemichannel function, and the expression of key proteins.Connexin hemichannel blockade improves survival of striatal GABA-ergic neurons after global cerebral ischaemia in term-equivalent fetal sheep.Mind the Gaps in Tumor Immunity: Impact of Connexin-Mediated Intercellular Connections.Gap junction intercellular communication mediated by connexin43 in astrocytes is essential for their resistance to oxidative stressThe action of mimetic peptides on connexins protects fibroblasts from the negative effects of ischemia reperfusionBoldine prevents renal alterations in diabetic rats.Evaluation of Connexin 43 Redistribution and Endocytosis in Astrocytes Subjected to Ischemia/Reperfusion or Oxygen-Glucose Deprivation and Reoxygenation.Connexin 43 hemichannels mediate the Ca2+ influx induced by extracellular alkalinization.Glucose increases intracellular free Ca(2+) in tanycytes via ATP released through connexin 43 hemichannels.Inhibition of Cx43 mediates protective effects on hypoxic/reoxygenated human neuroblastoma cellsHemichannel-mediated release of lactate.Acute connexin43 temporal and spatial expression in response to ischemic stroke.
P2860
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P2860
Hypoxia in high glucose followed by reoxygenation in normal glucose reduces the viability of cortical astrocytes through increased permeability of connexin 43 hemichannels.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@en
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@nl
type
label
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@en
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@nl
prefLabel
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@en
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@nl
P2093
P2860
P921
P356
P1433
P1476
Hypoxia in high glucose follow ...... y of connexin 43 hemichannels.
@en
P2093
Diego E Hernández
Juan A Orellana
Juan C Sáez
Michael V L Bennett
Victoria Velarde
P2860
P304
P356
10.1002/GLIA.20926
P577
2010-02-01T00:00:00Z