Ang II-salt hypertension depends on neuronal activity in the hypothalamic paraventricular nucleus but not on local actions of tumor necrosis factor-α.
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Coping with dehydration: sympathetic activation and regulation of glutamatergic transmission in the hypothalamic PVN.Acute cyclooxygenase inhibition does not alter muscle sympathetic nerve activity or forearm vasodilator responsiveness in lean and obese adults.Activation of corticotropin-releasing factor receptors in the rostral ventrolateral medulla is required for glucose-induced sympathoexcitation.Functional neural-bone marrow pathways: implications in hypertension and cardiovascular disease.Activation of the hypothalamic paraventricular nucleus by forebrain hypertonicity selectively increases tonic vasomotor sympathetic nerve activity.Hypothalamic signaling mechanisms in hypertension.Sympathoexcitation in ANG II-salt hypertension involves reduced SK channel function in the hypothalamic paraventricular nucleus.High Salt Intake Augments Excitability of PVN Neurons in Rats: Role of the Endoplasmic Reticulum Ca2+ StoreAngiotensin-II, the Brain, and Hypertension: An Update.Recent Advances in Neurogenic Hypertension: Dietary Salt, Obesity, and Inflammation.Ultrastructural characterization of tumor necrosis factor alpha receptor type 1 distribution in the hypothalamic paraventricular nucleus of the mouse.Update on angiotensin II: new endocrine connections between the brain, adrenal glands and the cardiovascular system.Central and Peripheral Slow-Pressor Mechanisms Contributing to.The Hypothalamic PVN Contributes to Acute Intermittent Hypoxia-Induced Sympathetic but not Phrenic Long-Term Facilitation.Burst Patterning of Hypothalamic Paraventricular Nucleus-Driven Sympathetic Nerve Activity in Angiotensin II-Salt Hypertension.Long-Term High Salt Intake Involves Reduced SK Currents and Increased Excitability of PVN Neurons with Projections to the Rostral Ventrolateral Medulla in Rats.Hypertension, TLR4 activation in brain and cardiac hypertrophy.Blood pressure is maintained during dehydration by hypothalamic paraventricular nucleus-driven tonic sympathetic nerve activity.Expression of Proinflammatory Cytokines Is Upregulated in the Hypothalamic Paraventricular Nucleus of Dahl Salt-Sensitive Hypertensive Rats.
P2860
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P2860
Ang II-salt hypertension depends on neuronal activity in the hypothalamic paraventricular nucleus but not on local actions of tumor necrosis factor-α.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@en
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@nl
type
label
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@en
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@nl
prefLabel
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@en
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@nl
P2093
P2860
P1433
P1476
Ang II-salt hypertension depen ...... ns of tumor necrosis factor-α.
@en
P2093
Glenn M Toney
Megan E Bardgett
Myrna Herrera-Rosales
Walter W Holbein
P2860
P304
P356
10.1161/HYPERTENSIONAHA.113.02429
P407
P577
2013-12-09T00:00:00Z