Residual Cdk1/2 activity after DNA damage promotes senescence.
about
How the cell cycle enforces senescence.Around and beyond 53BP1 Nuclear Bodies.Orchestration of DNA Damage Checkpoint Dynamics across the Human Cell Cycle.Human Cytomegalovirus Nuclear Capsids Associate with the Core Nuclear Egress Complex and the Viral Protein Kinase pUL97.Comprehensive analysis of differential expression profiles reveals potential biomarkers associated with the cell cycle and regulated by p53 in human small cell lung cancer.Ionizing Radiation-Induced Cellular Senescence in Normal, Non-transformed Cells and the Involved DNA Damage Response: A Mini Review.Persistent repair intermediates induce senescence
P2860
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P2860
Residual Cdk1/2 activity after DNA damage promotes senescence.
description
2017 nî lūn-bûn
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name
Residual Cdk1/2 activity after DNA damage promotes senescence.
@en
Residual Cdk1/2 activity after DNA damage promotes senescence.
@nl
type
label
Residual Cdk1/2 activity after DNA damage promotes senescence.
@en
Residual Cdk1/2 activity after DNA damage promotes senescence.
@nl
prefLabel
Residual Cdk1/2 activity after DNA damage promotes senescence.
@en
Residual Cdk1/2 activity after DNA damage promotes senescence.
@nl
P2093
P2860
P356
P1433
P1476
Residual Cdk1/2 activity after DNA damage promotes senescence
@en
P2093
Erik Müllers
Kamila Burdova
Libor Macurek
P2860
P304
P356
10.1111/ACEL.12588
P577
2017-03-26T00:00:00Z