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Developmental regulation of Th17-cell capacity in human neonates

Developmental regulation of Th17-cell capacity in human neonates

http://www.wikidata.org/entity/Q42025675

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Elucidation of pathways driving asthma pathogenesis: development of a systems-level analytic strategy Impact of In Utero Exposure to Malaria on Fetal T Cell Immunity.Vitamin D insufficiency in neonatal hypoxic-ischemic encephalopathy.Augmented Th17-type immune responses in preterm neonates exposed to histologic chorioamnionitis.Whole Blood Profiling of Bacillus Calmette-Guérin-Induced Trained Innate Immunity in Infants Identifies Epidermal Growth Factor, IL-6, Platelet-Derived Growth Factor-AB/BB, and Natural Killer Cell Activation.Unbalanced Neonatal CD4(+) T-Cell Immunity Ontogeny of myeloid cells.Endoplasmic reticulum stress, unfolded protein response and altered T cell differentiation in necrotizing enterocolitis Immune responses in neonates.What Causes Biliary Atresia? Unique Aspects of the Neonatal Immune System Provide Clues to Disease Pathogenesis Existence of Th22 in children and evaluation of IL-22 + CD4 + T, Th17, and other T cell effector subsets from healthy children compared to adults A Neonatal Murine Model of MRSA Pneumonia.Innate immune function by Toll-like receptors: distinct responses in newborns and the elderly.Lipopolysaccharide-Induced Chorioamnionitis Promotes IL-1-Dependent Inflammatory FOXP3+ CD4+ T Cells in the Fetal Rhesus Macaque.Intra-amniotic IL-1β induces fetal inflammation in rhesus monkeys and alters the regulatory T cell/IL-17 balance Immunology of pediatric HIV infection Early pediatric atopic dermatitis shows only a cutaneous lymphocyte antigen (CLA)(+) TH2/TH1 cell imbalance, whereas adults acquire CLA(+) TH22/TC22 cell subsets Neonatal immunology: responses to pathogenic microorganisms and epigenetics reveal an "immunodiverse" developmental state.Maternal inflammation modulates infant immune response patterns to viral lung challenge in a murine model.Both inflammatory and regulatory cytokine responses to malaria are blunted with increasing age in highly exposed children.Neonatal neutrophils stimulated by group B Streptococcus induce a proinflammatory T-helper cell bias.Newborn susceptibility to infection vs. disease depends on complex in vivo interactions of host and pathogen.Factors Affecting the Immunity to Respiratory Syncytial Virus: From Epigenetics to Microbiome.Transcriptome analysis of immune genes in peripheral blood mononuclear cells of young foals and adult horses

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Developmental regulation of Th17-cell capacity in human neonates

http://www.wikidata.org/entity/Q42025675

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

@zh-tw

2011年论文

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2011年论文

@zh

2011年论文

@zh-cn

name

Developmental regulation of Th17-cell capacity in human neonates

@en

Developmental regulation of Th17-cell capacity in human neonates

@nl

type

label

Developmental regulation of Th17-cell capacity in human neonates

@en

Developmental regulation of Th17-cell capacity in human neonates

@nl

prefLabel

Developmental regulation of Th17-cell capacity in human neonates

@en

Developmental regulation of Th17-cell capacity in human neonates

@nl

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European Journal of Immunology

P1476

Developmental regulation of Th17-cell capacity in human neonates

@en

P2093

Allison Black

http://www.w3.org/2001/XMLSchema#string

David A Randolph

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Richard L Kirkman

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Suniti Bhaumik

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P2860

The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

Transforming growth factor-beta induces development of the T(H)17 lineage

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Impaired T(H)17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

Differentiation of effector CD4 T cell populations (*)

IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways

IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

Development, cytokine profile and function of human interleukin 17-producing helper T cells

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311-319

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scholarly article

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10.1002/EJI.201141847

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2

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42

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Casey T. Weaver

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2011-12-19T00:00:00Z

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51816093

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22101893

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3414367

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