Avoiding chromosome pathology when replication forks collide.
about
Replication Termination: Containing Fork Fusion-Mediated Pathologies in Escherichia coliNew Insights into the Functions of Transcription Factors that Bind the RNA Polymerase Secondary ChannelRibonucleotides in bacterial DNADifferential management of the replication terminus regions of the two Vibrio cholerae chromosomes during cell divisionChromosomal Replication Complexity: A Novel DNA Metrics and Genome Instability FactorLinear mtDNA fragments and unusual mtDNA rearrangements associated with pathological deficiency of MGME1 exonuclease.Recombination and replication.Completion of DNA replication in Escherichia coli.Roles of type 1A topoisomerases in genome maintenance in Escherichia coli.Branch migration prevents DNA loss during double-strand break repairFunctions that protect Escherichia coli from DNA-protein crosslinks.End of the beginning: elongation and termination features of alternative modes of chromosomal replication initiation in bacteriaRECG maintains plastid and mitochondrial genome stability by suppressing extensive recombination between short dispersed repeatsDirected Evolution of RecA Variants with Enhanced Capacity for Conjugational RecombinationRecBCD is required to complete chromosomal replication: Implications for double-strand break frequencies and repair mechanismsRecG Directs DNA Synthesis during Double-Strand Break Repair.Quantitative genomic analysis of RecA protein binding during DNA double-strand break repair reveals RecBCD action in vivoThe progression of replication forks at natural replication barriers in live bacteriaLong range chromosome organization in Escherichia coli: The position of the replication origin defines the non-structured regions and the Right and Left macrodomains.Replication of the Escherichia coli chromosome in RNase HI-deficient cells: multiple initiation regions and fork dynamics.Chromosome replication origins: do we really need them?Archaeal chromosome biology.A Replisome's journey through the bacterial chromosome.RecG controls DNA amplification at double-strand breaks and arrested replication forks.Replication Restart in Bacteria.Mechanisms of DNA replication termination.Cellular location and activity of Escherichia coli RecG proteins shed light on the function of its structurally unresolved C-terminus.25 years on and no end in sight: a perspective on the role of RecG proteinDifferent genome stability proteins underpin primed and naïve adaptation in E. coli CRISPR-Cas immunity.The Consequences of Replicating in the Wrong Orientation: Bacterial Chromosome Duplication without an Active Replication OriginLambda gpP-DnaB Helicase Sequestration and gpP-RpoB Associated Effects: On Screens for Auxotrophs, Selection for Rif(R), Toxicity, Mutagenicity, Plasmid Curing.Shaping the landscape of the Escherichia coli chromosome: replication-transcription encounters in cells with an ectopic replication origin.Escherichia coli RadD Protein Functionally Interacts with the Single-stranded DNA-binding Protein.High-resolution replication profiles define the stochastic nature of genome replication initiation and termination.The dynamics of genome replication using deep sequencing.Termination of DNA replication forks: "Breaking up is hard to do".Division-induced DNA double strand breaks in the chromosome terminus region of Escherichia coli lacking RecBCD DNA repair enzyme.The mechanism of DNA replication termination in vertebrates.Mechanisms of bacterial DNA replication restart.The RECG1 DNA Translocase Is a Key Factor in Recombination Surveillance, Repair, and Segregation of the Mitochondrial DNA in Arabidopsis.
P2860
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P2860
Avoiding chromosome pathology when replication forks collide.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Avoiding chromosome pathology when replication forks collide.
@en
Avoiding chromosome pathology when replication forks collide.
@nl
type
label
Avoiding chromosome pathology when replication forks collide.
@en
Avoiding chromosome pathology when replication forks collide.
@nl
prefLabel
Avoiding chromosome pathology when replication forks collide.
@en
Avoiding chromosome pathology when replication forks collide.
@nl
P2093
P2860
P50
P356
P1433
P1476
Avoiding chromosome pathology when replication forks collide.
@en
P2093
Amy L Upton
Anna Stockum
Christian J Rudolph
Conrad A Nieduszynski
P2860
P2888
P304
P356
10.1038/NATURE12312
P407
P577
2013-07-28T00:00:00Z
P5875
P6179
1011222140