Avoiding chromosome pathology when replication forks collide. - Wikidata - wikimedia - TriplyDB

Avoiding chromosome pathology when replication forks collide.

Avoiding chromosome pathology when replication forks collide.

http://www.wikidata.org/entity/Q42032142

about

Replication Termination: Containing Fork Fusion-Mediated Pathologies in Escherichia coli New Insights into the Functions of Transcription Factors that Bind the RNA Polymerase Secondary Channel Ribonucleotides in bacterial DNA Differential management of the replication terminus regions of the two Vibrio cholerae chromosomes during cell division Chromosomal Replication Complexity: A Novel DNA Metrics and Genome Instability Factor Linear mtDNA fragments and unusual mtDNA rearrangements associated with pathological deficiency of MGME1 exonuclease.Recombination and replication.Completion of DNA replication in Escherichia coli.Roles of type 1A topoisomerases in genome maintenance in Escherichia coli.Branch migration prevents DNA loss during double-strand break repair Functions that protect Escherichia coli from DNA-protein crosslinks.End of the beginning: elongation and termination features of alternative modes of chromosomal replication initiation in bacteria RECG maintains plastid and mitochondrial genome stability by suppressing extensive recombination between short dispersed repeats Directed Evolution of RecA Variants with Enhanced Capacity for Conjugational Recombination RecBCD is required to complete chromosomal replication: Implications for double-strand break frequencies and repair mechanisms RecG Directs DNA Synthesis during Double-Strand Break Repair.Quantitative genomic analysis of RecA protein binding during DNA double-strand break repair reveals RecBCD action in vivo The progression of replication forks at natural replication barriers in live bacteria Long range chromosome organization in Escherichia coli: The position of the replication origin defines the non-structured regions and the Right and Left macrodomains.Replication of the Escherichia coli chromosome in RNase HI-deficient cells: multiple initiation regions and fork dynamics.Chromosome replication origins: do we really need them?Archaeal chromosome biology.A Replisome's journey through the bacterial chromosome.RecG controls DNA amplification at double-strand breaks and arrested replication forks.Replication Restart in Bacteria.Mechanisms of DNA replication termination.Cellular location and activity of Escherichia coli RecG proteins shed light on the function of its structurally unresolved C-terminus.25 years on and no end in sight: a perspective on the role of RecG protein Different genome stability proteins underpin primed and naïve adaptation in E. coli CRISPR-Cas immunity.The Consequences of Replicating in the Wrong Orientation: Bacterial Chromosome Duplication without an Active Replication Origin Lambda gpP-DnaB Helicase Sequestration and gpP-RpoB Associated Effects: On Screens for Auxotrophs, Selection for Rif(R), Toxicity, Mutagenicity, Plasmid Curing.Shaping the landscape of the Escherichia coli chromosome: replication-transcription encounters in cells with an ectopic replication origin.Escherichia coli RadD Protein Functionally Interacts with the Single-stranded DNA-binding Protein.High-resolution replication profiles define the stochastic nature of genome replication initiation and termination.The dynamics of genome replication using deep sequencing.Termination of DNA replication forks: "Breaking up is hard to do".Division-induced DNA double strand breaks in the chromosome terminus region of Escherichia coli lacking RecBCD DNA repair enzyme.The mechanism of DNA replication termination in vertebrates.Mechanisms of bacterial DNA replication restart.The RECG1 DNA Translocase Is a Key Factor in Recombination Surveillance, Repair, and Segregation of the Mitochondrial DNA in Arabidopsis.

P2860

Q26741314-84F83A3B-6074-4209-8999-1AEC1AEFB410 Q26822940-D21DD708-E784-4DD1-9E53-D5D7F11165F7 Q26864225-F2C2EFE7-03FF-4BC0-B309-229278AAAA04 Q27314157-568D2DC7-35AB-474F-912B-7C3EDB9A290B Q28078118-4CE0F5A0-E6B2-4986-ABD2-134D93C3FAB1 Q34427479-5579F501-8A10-4B71-ACB2-C9AE7F328553 Q34444668-1C6B3A5D-7EAC-4E21-9545-7E324848DB4A Q34581032-7B8AE1FB-3CA5-4E68-B0B1-A94FC9FE6958 Q35221111-E69C8346-D6EB-4A18-85C1-2A5594F62175 Q35221144-D568D3E1-7613-4F23-A134-849C7C716B00 Q35268535-EFE1234E-0C78-4C74-BAB1-54B3B146D77D Q35539851-ACB0D3A5-39FD-4CBD-A256-0292D0F36058 Q35577068-3B142854-94B3-430E-8532-E97BC0C9A740 Q35654604-1B666B40-04F1-477F-9A5A-21E8FD9EB646 Q35908909-D90A9684-A205-4C2F-82DF-763DCFBBFFE1 Q35922661-775FBBC8-FB60-43B1-9CCC-57AB5EDB3747 Q36008202-7502A7A2-1E5B-4D8D-AB56-D6F563CF8566 Q36013967-B0768437-2167-4B1F-9910-CFEE28EBABDF Q36366966-1791C764-00DA-4FEB-B634-62941E7A82DA Q37588219-960EDDB0-4701-4B37-A050-B1C8F530C2D5 Q38197136-3E26ED86-D10A-4CA6-A58B-E9A8032F8A16 Q38366852-0D7C0DBB-4A77-4F20-9C19-2E1392315D2B Q38534013-9A5F4995-A72D-44DE-91DA-4CCD7B055904 Q39117161-EF5BB017-51FA-44A3-8383-9B5DA49773F6 Q39188771-642AF033-0660-4EF3-B636-16391282E031 Q39326104-FF854D9D-2B14-4B3A-AF90-E97F67F39BD3 Q39769826-09CFC085-37B7-4F4A-B7CA-53D7A5D95DFD Q39869214-BCE5F780-1904-49BD-8FE7-0FEB3957DB35 Q40310719-2630E4C9-6BBC-491E-8D47-BB1E0F48596F Q40360421-E9639CE1-E356-46B5-B378-3CCA0BABBBB8 Q40684824-93AD92F7-4A3E-4482-ACD0-06BECFC99596 Q40752063-D2F6D259-4F40-419F-BDDE-CAD886D424F1 Q41161859-5EB139E0-E276-472D-9614-C7B7720280C3 Q41885231-8A2FFE36-F157-4C12-AC08-1E681FE60356 Q42090523-17665A82-681F-401E-8EF4-72D793D558CF Q42276650-4AF30D78-796B-4EB3-AC17-F6AA581D0583 Q42652312-D0329542-E48A-4AAF-B7B3-0774C5FA2185 Q43245960-2D8B4C8A-648F-4922-9F57-9A444666B819 Q47334226-C932C662-EFCB-4158-AC25-9C070C86ED9D Q48120447-DCBEF7CC-5DDD-4E26-8EF1-C0C0491EAD26

P2860

Avoiding chromosome pathology when replication forks collide.

http://www.wikidata.org/entity/Q42032142

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

Avoiding chromosome pathology when replication forks collide.

@en

Avoiding chromosome pathology when replication forks collide.

@nl

type

label

Avoiding chromosome pathology when replication forks collide.

@en

Avoiding chromosome pathology when replication forks collide.

@nl

prefLabel

Avoiding chromosome pathology when replication forks collide.

@en

Avoiding chromosome pathology when replication forks collide.

@nl

P1433

Q42032142-BB8DFC99-6454-4822-8D44-FB28A246B73B

P1476

Q42032142-B87F5CF4-91FD-494B-9F80-8296E478A131

P2093

Q42032142-111DEE32-157B-497C-9491-A0E840E04A5F

Q42032142-25F4FCB5-F755-42ED-A896-83BC340ACD45

Q42032142-658360FF-4A73-4073-B1A5-1BF2B1F55B53

Q42032142-6B18AC4A-0414-42DD-AA4F-5CB67A13DCE3

P2860

Q42032142-0B80D712-88AC-412A-B5D6-13100196E867

Q42032142-0D83CDD3-71B8-4D17-ABD1-4226F7B9B266

Q42032142-17BA9737-C9D2-4655-A9CA-E17DAB0EB1C3

Q42032142-2A012DD2-B03B-42F4-84CA-77AD87C23E3E

Q42032142-2B3E4172-67DA-47D0-8694-2E5B96BF06B0

Q42032142-2F939B1A-0A88-4086-A9F2-7128699748A3

Q42032142-31571C9D-1623-4417-B4DC-FEF6C123F0B6

Q42032142-327BE964-AE8D-4814-A0FB-F9AE392EA15A

Q42032142-5DF0C468-25B1-4216-9116-C7F1E4118BB5

Q42032142-680C9ABD-F803-4270-A59C-3C161E93C399

P2888

Q42032142-580DD498-24A0-40E0-8711-EC82105E0AA0

P304

Q42032142-D05BF386-6BB4-47C7-AB0A-9B1C77F03BC2

P31

Q42032142-59C452DA-5729-4BC3-996F-5D40DD5B874F

P356

Q42032142-52B19A95-2A1A-42ED-847A-C8EDE6F6F75B

P407

Q42032142-B98EA362-6A49-4F2C-91DE-FF56E92EF9E5

P433

Q42032142-12758ACB-09C3-4359-8895-BD64A9837272

P478

Q42032142-6A4899B6-4990-402C-910A-FE34FA41098F

P50

Q42032142-1B6F1DD7-8A93-48C1-94CF-C41BD1F41662

Q42032142-B0D9FB7B-5E37-43E2-900E-C09F4A4D4907

Q42032142-CB7E74C4-80C9-4D98-A27F-D51D82910FDF

P577

Q42032142-D20B174E-557A-43CB-972E-4741A72DDF63

P5875

Q42032142-3355F829-1622-4980-B5AA-85D6944C84E0

P6179

Q42032142-6B0F342F-3AE9-4B00-B1A0-EEF5FC978E67

P698

Q42032142-5858C74D-D094-4DAC-AAEC-4DAF772F5DDD

P921

Q42032142-772be713-03c5-4e8b-9083-cda51da60c81

P932

Q42032142-BF0BE620-F844-4E03-AD69-78A818C02B18

P356

P1433

P1476

Avoiding chromosome pathology when replication forks collide.

@en

P2093

Amy L Upton

http://www.w3.org/2001/XMLSchema#string

Anna Stockum

http://www.w3.org/2001/XMLSchema#string

Christian J Rudolph

http://www.w3.org/2001/XMLSchema#string

Conrad A Nieduszynski

http://www.w3.org/2001/XMLSchema#string

P2860

Loss-of-function mutations in MGME1 impair mtDNA replication and cause multisystemic mitochondrial disease

One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products

Replication termination at eukaryotic chromosomes is mediated by Top2 and occurs at genomic loci containing pausing elements

Initiation of genetic recombination and recombination-dependent replication

Replication and segregation of an Escherichia coli chromosome with two replication origins.

The plasmid prophage N15: a linear DNA with covalently closed ends.

Escherichia coli RecG functionally suppresses human Bloom syndrome phenotypes

DNA replication initiation: mechanisms and regulation in bacteria.

Genome-wide detection of chromosomal rearrangements, indels, and mutations in circular chromosomes by short read sequencing

Escherichia coli with a linear genome

P2888

P304

608-611

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/NATURE12312

http://www.w3.org/2001/XMLSchema#string

P407

P433

7464

http://www.w3.org/2001/XMLSchema#string

P478

500

http://www.w3.org/2001/XMLSchema#string

P50

Conrad A. Nieduszynski

Christian J Rudolph

Robert G. Lloyd

P577

2013-07-28T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

253334777

http://www.w3.org/2001/XMLSchema#string

P6179

1011222140

http://www.w3.org/2001/XMLSchema#string

P698

23892781

http://www.w3.org/2001/XMLSchema#string

P921

P932

3819906

http://www.w3.org/2001/XMLSchema#string