Heparin-binding epidermal growth factor-like growth factor (HB-EGF) is increased in osteoarthritis and regulates chondrocyte catabolic and anabolic activities

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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Heparin-binding epidermal grow ...... abolic and anabolic activities

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Heparin-binding epidermal grow ...... abolic and anabolic activities

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Heparin-binding epidermal grow ...... abolic and anabolic activities

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P1476

Heparin-binding epidermal grow ...... abolic and anabolic activities

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P2093

D L Long

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R F Loeser

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S Chubinskaya

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V Ulici

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P2860

Cloning, expression, and type II collagenolytic activity of matrix metalloproteinase-13 from human osteoarthritic cartilage

Opposing BMP and EGF signalling pathways converge on the TGF-beta family mediator Smad1

Fibronectin fragment activation of proline-rich tyrosine kinase PYK2 mediates integrin signals regulating collagenase-3 expression by human chondrocytes through a protein kinase C-dependent pathway

Integration of gene expression data with network-based analysis to identify signaling and metabolic pathways regulated during the development of osteoarthritis

Oxidative stress inhibits insulin-like growth factor-I induction of chondrocyte proteoglycan synthesis through differential regulation of phosphatidylinositol 3-Kinase-Akt and MEK-ERK MAPK signaling pathways.

Gene expression pattern of cells from inflamed and normal areas of osteoarthritis synovial membrane

Matrix metalloproteinase 13-deficient mice are resistant to osteoarthritic cartilage erosion but not chondrocyte hypertrophy or osteophyte development.

Targeted disruption of Mig-6 in the mouse genome leads to early onset degenerative joint disease.

NF-kappa B mediates the stimulation of cytokine and chemokine expression by human articular chondrocytes in response to fibronectin fragments

Osteoarthritis: a disease of the joint as an organ.

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scholarly article

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10.1016/J.JOCA.2015.04.019

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2015-04-29T00:00:00Z

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