The microRNA-26a target E2F7 sustains cell proliferation and inhibits monocytic differentiation of acute myeloid leukemia cells.
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The Potential of Vitamin D-Regulated Intracellular Signaling Pathways as Targets for Myeloid Leukemia TherapyMicroRNA-regulated pathways in hematological malignancies: how to avoid cells playing out of tuneArsenic responsive microRNAs in vivo and their potential involvement in arsenic-induced oxidative stressMicroRNA-26a-5p and microRNA-23b-3p up-regulate peroxiredoxin III in acute myeloid leukemia.PU.1-Regulated Long Noncoding RNA lnc-MC Controls Human Monocyte/Macrophage Differentiation through Interaction with MicroRNA 199a-5p.MiR-26a functions oppositely in osteogenic differentiation of BMSCs and ADSCs depending on distinct activation and roles of Wnt and BMP signaling pathway.Loss of connective tissue growth factor as an unfavorable prognosis factor activates miR-18b by PI3K/AKT/C-Jun and C-Myc and promotes cell growth in nasopharyngeal carcinomaE2F7 regulates transcription and maturation of multiple microRNAs to restrain cell proliferationArgonaute 2 sustains the gene expression program driving human monocytic differentiation of acute myeloid leukemia cells.High Expression of CPT1A Predicts Adverse Outcomes: A Potential Therapeutic Target for Acute Myeloid Leukemia.Low 25(OH) vitamin D3 levels are associated with adverse outcome in newly diagnosed, intensively treated adult acute myeloid leukemia.Synergistic functions of E2F7 and E2F8 are critical to suppress stress-induced skin cancer.Pontin is a critical regulator for AML1-ETO-induced leukemia.E2F Transcription Factors Control the Roller Coaster Ride of Cell Cycle Gene Expression.MicroRNA: an important regulator in acute myeloid leukemia.MiR-26a inhibits stem cell-like phenotype and tumor growth of osteosarcoma by targeting Jagged1.The role of microRNA-26a in human cancer progression and clinical application.New insights into vitamin D anticancer properties: focus on miRNA modulation.The miR-223 host non-coding transcript linc-223 induces IRF4 expression in acute myeloid leukemia by acting as a competing endogenous RNA.Effect of miR-204&211 and RUNX2 control on the fate of human mesenchymal stromal cells.microRNAs and Acute Myeloid Leukemia Chemoresistance: A Mechanistic Overview.[MicroRNA-26a and Tumor].Role of MicroRNAs in the development and function of innate immune cells.MicroRNA-30a-5p inhibits gallbladder cancer cell proliferation, migration and metastasis by targeting E2F7.microRNA-26a induces a mitochondrial apoptosis mediated by p53 through targeting to inhibit Mcl1 in human hepatocellular carcinoma.
P2860
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P2860
The microRNA-26a target E2F7 sustains cell proliferation and inhibits monocytic differentiation of acute myeloid leukemia cells.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
The microRNA-26a target E2F7 s ...... acute myeloid leukemia cells.
@en
The microRNA-26a target E2F7 s ...... acute myeloid leukemia cells.
@nl
type
label
The microRNA-26a target E2F7 s ...... acute myeloid leukemia cells.
@en
The microRNA-26a target E2F7 s ...... acute myeloid leukemia cells.
@nl
prefLabel
The microRNA-26a target E2F7 s ...... acute myeloid leukemia cells.
@en
The microRNA-26a target E2F7 s ...... acute myeloid leukemia cells.
@nl
P2093
P2860
P356
P1476
The microRNA-26a target E2F7 s ...... f acute myeloid leukemia cells
@en
P2093
A Mangiavacchi
B Salvatori
N Peragine
S Chiaretti
P2860
P356
10.1038/CDDIS.2012.151
P577
2012-10-25T00:00:00Z