Is left-right asymmetry a form of planar cell polarity? - Wikidata - wikimedia - TriplyDB

Is left-right asymmetry a form of planar cell polarity?

Is left-right asymmetry a form of planar cell polarity?

http://www.wikidata.org/entity/Q42067932

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Left-right determination: involvement of molecular motor KIF3, cilia, and nodal flow Endogenous voltage gradients as mediators of cell-cell communication: strategies for investigating bioelectrical signals during pattern formation A unified model for left-right asymmetry? Comparison and synthesis of molecular models of embryonic laterality Ciliary proteins Bbs8 and Ift20 promote planar cell polarity in the cochlea Rapid differential transport of Nodal and Lefty on sulfated proteoglycan-rich extracellular matrix regulates left-right asymmetry in Xenopus Low frequency vibrations disrupt left-right patterning in the Xenopus embryo The ATP-sensitive K(+)-channel (K(ATP)) controls early left-right patterning in Xenopus and chick embryos.Left-right symmetry breaking in tissue morphogenesis via cytoskeletal mechanics.Histone deacetylase activity is necessary for left-right patterning during vertebrate development Long-range neural and gap junction protein-mediated cues control polarity during planarian regeneration.Early, nonciliary role for microtubule proteins in left-right patterning is conserved across kingdoms.Molecular bioelectricity: how endogenous voltage potentials control cell behavior and instruct pattern regulation in vivo.Serotonin has early, cilia-independent roles in Xenopus left-right patterning.Left-right symmetry breaking in mice by left-right dynein may occur via a biased chromatid segregation mechanism, without directly involving the Nodal gene.Micropatterning of cells reveals chiral morphogenesis.Primary cilia and kidney injury: current research status and future perspectives Reprogramming cells and tissue patterning via bioelectrical pathways: molecular mechanisms and biomedical opportunities Bioelectric mechanisms in regeneration: Unique aspects and future perspectives.It's never too early to get it Right: A conserved role for the cytoskeleton in left-right asymmetry.Left-right asymmetry in gut development: what happens next?The role of proton transporters in epithelial Wnt signaling pathways.Left-right patterning in the C. elegans embryo: Unique mechanisms and common principles.Getting to the heart of planar cell polarity signaling.Molecular bioelectricity in developmental biology: new tools and recent discoveries: control of cell behavior and pattern formation by transmembrane potential gradients.Planar cell polarity signaling: coordination of cellular orientation across tissues.Right across the tree of life: the evolution of left-right asymmetry in the Bilateria.Regulation of basal body and ciliary functions by Diversin.Consistent left-right asymmetry cannot be established by late organizers in Xenopus unless the late organizer is a conjoined twin.Left-right asymmetry in the chick embryo requires core planar cell polarity protein Vangl2.Polarity proteins are required for left-right axis orientation and twin-twin instruction.Bioelectric signaling in regeneration: Mechanisms of ionic controls of growth and form.

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P2860

Is left-right asymmetry a form of planar cell polarity?

http://www.wikidata.org/entity/Q42067932

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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name

Is left-right asymmetry a form of planar cell polarity?

@en

Is left-right asymmetry a form of planar cell polarity?

@nl

type

label

Is left-right asymmetry a form of planar cell polarity?

@en

Is left-right asymmetry a form of planar cell polarity?

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prefLabel

Is left-right asymmetry a form of planar cell polarity?

@en

Is left-right asymmetry a form of planar cell polarity?

@nl

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Is left-right asymmetry a form of planar cell polarity?

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P2093

Michael Levin

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P2860

The mother centriole plays an instructive role in defining cell geometry

Randomization of left-right asymmetry due to loss of nodal cilia generating leftward flow of extraembryonic fluid in mice lacking KIF3B motor protein

Basal body dysfunction is a likely cause of pleiotropic Bardet-Biedl syndrome

Bardet-Biedl syndrome genes are important in retrograde intracellular trafficking and Kupffer's vesicle cilia function

Loss of nephrocystin-3 function can cause embryonic lethality, Meckel-Gruber-like syndrome, situs inversus, and renal-hepatic-pancreatic dysplasia

The Bardet-Biedl protein BBS4 targets cargo to the pericentriolar region and is required for microtubule anchoring and cell cycle progression

Inversin forms a complex with catenins and N-cadherin in polarized epithelial cells.

Polaris, a protein involved in left-right axis patterning, localizes to basal bodies and cilia

The ankyrin repeat protein Diversin recruits Casein kinase Iepsilon to the beta-catenin degradation complex and acts in both canonical Wnt and Wnt/JNK signaling

Frizzled6 controls hair patterning in mice

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355-366

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10.1242/DEV.015974

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