Spontaneous periodontitis development in diabetic rats involves an unrestricted expression of inflammatory cytokines and tissue destructive factors in the absence of major changes in commensal oral microbiota.

Spontaneous periodontitis development in diabetic rats involves an unrestricted expression of inflammatory cytokines and tissue destructive factors in the absence of major changes in commensal oral microbiota.

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http://www.wikidata.org/entity/Q42073506
Assessment of Alloxan-Induced Diabetic Rats as a Periodontal Disease Model Using a Selective Cyclooxygenase (COX)-2 InhibitorRole of the gastrointestinal ecosystem in the development of type 1 diabetes.Green Tea Modulates Cytokine Expression in the Periodontium and Attenuates Alveolar Bone Resorption in Type 1 Diabetic Rats.4-methoxycarbonyl curcumin: a unique inhibitor of both inflammatory mediators and periodontal inflammationA review of the evidence for pathogenic mechanisms that may link periodontitis and diabetes.Effect of high glucose, Porphyromonas gingivalis lipopolysaccharide and advanced glycation end-products on production of interleukin-6/-8 by gingival fibroblasts.Cell culture conditions: from outer space-like conditions to the mimicking of complex in vivo environments.A review of the evidence for pathogenic mechanisms that may link periodontitis and diabetes.Ligature-associated bacterial profiles are linked to type 2 diabetes mellitus in a rat model and influenced by antibody treatment against TNF-α or RAGE.A Chemically Modified Curcumin (CMC 2.24) Inhibits Nuclear Factor κB Activation and Inflammatory Bone Loss in Murine Models of LPS-Induced Experimental Periodontitis and Diabetes-Associated Natural Periodontitis.A novel chemically modified curcumin reduces inflammation-mediated connective tissue breakdown in a rat model of diabetes: periodontal and systemic effects.Importance of Mechanical Periodontal Therapy in Patients with Diabetes Type 2 and Periodontitis
P2860

Spontaneous periodontitis development in diabetic rats involves an unrestricted expression of inflammatory cytokines and tissue destructive factors in the absence of major changes in commensal oral microbiota.

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http://www.wikidata.org/entity/Q42073506
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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Spontaneous periodontitis deve ...... in commensal oral microbiota.
@en
Spontaneous periodontitis deve ...... in commensal oral microbiota.
@nl
type
label
Spontaneous periodontitis deve ...... in commensal oral microbiota.
@en
Spontaneous periodontitis deve ...... in commensal oral microbiota.
@nl
prefLabel
Spontaneous periodontitis deve ...... in commensal oral microbiota.
@en
Spontaneous periodontitis deve ...... in commensal oral microbiota.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P478
P577
P5875
P698
P921
P932
P356
P1433
P1476
Spontaneous periodontitis deve ...... in commensal oral microbiota.
@en
P2093
P2860
P304
P31
P356
P478
P577
P5875
P698
P921
P932