Pulmonary macrophages are attracted to inhaled particles through complement activation.
about
Selected new developments in asbestos immunotoxicityRespiratory health hazards in agricultureNanotoxicology: an emerging discipline evolving from studies of ultrafine particlesCritical role of MARCO in crystalline silica-induced pulmonary inflammationUp-regulated expression of transforming growth factor-alpha in the bronchiolar-alveolar duct regions of asbestos-exposed ratsRelation between pulmonary clearance and particle burden: a Michaelis-Menten-like kinetic modelChrysotile asbestos upregulates gene expression and production of alpha-receptors for platelet-derived growth factor (PDGF-AA) on rat lung fibroblasts.Morphological aspects of particle uptake by lung phagocytes.Pulmonary and thoracic macrophage subpopulations and clearance of particles from the lung.Role of the alveolar macrophage in lung injury: studies with ultrafine particles.Transmigration and phagocytosis of macrophages in an airway infection model using four-dimensional techniques.Functional consequences for primary human alveolar macrophages following treatment with long, but not short, multiwalled carbon nanotubes.Incorporation of tritiated thymidine by epithelial and interstitial cells in bronchiolar-alveolar regions of asbestos-exposed rats.Immunologic aspects of pneumoconiosis.The role of surfactant in the pulmonary reaction to mineral particles.In vivo particle uptake by airway macrophages in healthy volunteers.Interspecies comparisons of lung responses to inhaled particles and gases.Current perspectives on particulate induced pulmonary tumours.Persistent biological reactivity of quartz in the lung: raised protease burden compared with a non-pathogenic mineral dust and microbial particles.Species comparisons of proximal alveolar deposition patterns of inhaled particulates.Particle distribution in lung and lymph node tissues of rats and dogs and the migration of particle-containing alveolar cells in vitro.Mathematical modeling of the retention and clearance of low-toxicity particles in the lung.Migratory behaviors of alveolar macrophages during the alveolar clearance of light to heavy burdens of particles.Inorganic Particles Induce Secretion of a Macrophage Homologue of Platelet-Derived Growth Factor in a Density- and Time-Dependent Manner in VitroLung Dosimetry: Pulmonary Clearance of Inhaled Particles
P2860
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P2860
Pulmonary macrophages are attracted to inhaled particles through complement activation.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
@wuu
1988年学术文章
@zh-cn
1988年学术文章
@zh-hans
1988年学术文章
@zh-my
1988年学术文章
@zh-sg
1988年學術文章
@yue
1988年學術文章
@zh
1988年學術文章
@zh-hant
name
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@en
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@nl
type
label
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@en
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@nl
prefLabel
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@en
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@nl
P2093
P2860
P1476
Pulmonary macrophages are attracted to inhaled particles through complement activation.
@en
P2093
P2860
P356
10.3109/01902148809062850
P577
1988-01-01T00:00:00Z