Delayed circulatory failure due to the induction of nitric oxide synthase by lipoteichoic acid from Staphylococcus aureus in anaesthetized rats.
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Effects of tyrphostins and genistein on the circulatory failure and organ dysfunction caused by endotoxin in the rat: a possible role for protein tyrosine kinaseThe cell wall components peptidoglycan and lipoteichoic acid from Staphylococcus aureus act in synergy to cause shock and multiple organ failureCommercial preparations of lipoteichoic acid contain endotoxin that contributes to activation of mouse macrophages in vitroLipoteichoic acid from Staphylococcus aureus induces lung endothelial cell barrier dysfunction: role of reactive oxygen and nitrogen speciesStructure-function relationship of cytokine induction by lipoteichoic acid from Staphylococcus aureus.Over-expression of GTP-cyclohydrolase 1 feedback regulatory protein attenuates LPS and cytokine-stimulated nitric oxide production.Receptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.Endothelial nitric oxide synthase deficient mice are protected from lipopolysaccharide induced acute lung injury.Inhibition of the activity of poly(ADP ribose) synthetase reduces ischemia-reperfusion injury in the heart and skeletal muscle.Induction of nitric oxide production by polyosides from the cell walls of Streptococcus mutans OMZ 175, a gram-positive bacterium, in the rat aortaLipoteichoic acid induces nuclear factor-kappaB activation and nitric oxide synthase expression via phosphatidylinositol 3-kinase, Akt, and p38 MAPK in RAW 264.7 macrophages.Induction of nitric oxide synthase in RAW 264.7 macrophages by lipoteichoic acid from Staphylococcus aureus: involvement of protein kinase C- and nuclear factor-kB-dependent mechanisms.Mechanism of gram-positive shock: identification of peptidoglycan and lipoteichoic acid moieties essential in the induction of nitric oxide synthase, shock, and multiple organ failure.Analysis of the signal transduction in the induction of nitric oxide synthase by lipoteichoic acid in macrophagesRole of microbial proteases in pathogenesis.Effect of selective blockade of endothelin ETB receptors on the liver dysfunction and injury caused by endotoxaemia in the ratRole of nitric oxide in the circulatory failure and organ injury in a rodent model of gram-positive shockRole for intracellular platelet-activating factor in the circulatory failure in a model of gram-positive shock.Lipoteichoic acid modulates inflammatory response in macrophages after phagocytosis of titanium particles through Toll-like receptor 2 cascade and inflammasomes.
P2860
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P2860
Delayed circulatory failure due to the induction of nitric oxide synthase by lipoteichoic acid from Staphylococcus aureus in anaesthetized rats.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@en
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@nl
type
label
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@en
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@nl
prefLabel
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@en
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@nl
P2093
P2860
P1476
Delayed circulatory failure du ...... aureus in anaesthetized rats.
@en
P2093
C E Bryant
C Thiemermann
M L Hunter
S J De Kimpe
P2860
P304
P356
10.1111/J.1476-5381.1995.TB13349.X
P407
P577
1995-03-01T00:00:00Z