Primary cell model for activation-inducible human immunodeficiency virus
about
Recent developments in human immunodeficiency virus-1 latency researchDirectly infected resting CD4+T cells can produce HIV Gag without spreading infection in a model of HIV latencySmall-molecule screening using a human primary cell model of HIV latency identifies compounds that reverse latency without cellular activationCombinatorial signals from CD28 differentially regulate human immunodeficiency virus transcription in T cellsHIV persistence: chemokines and their signalling pathways.Combinatorial latency reactivation for HIV-1 subtypes and variantsCombination of biological screening in a cellular model of viral latency and virtual screening identifies novel compounds that reactivate HIV-1A flexible model of HIV-1 latency permitting evaluation of many primary CD4 T-cell reservoirs.The molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit.Towards an HIV cure: a global scientific strategyDisruption of Type I Interferon Induction by HIV Infection of T Cells.Human immunodeficiency virus (HIV) latency: the major hurdle in HIV eradicationHIV reservoirs and latency models.High levels of CD2 expression identify HIV-1 latently infected resting memory CD4+ T cells in virally suppressed subjects.Eradication of HIV: current challenges and new directions.HDAC inhibitors in HIV.Cellular and molecular mechanisms involved in the establishment of HIV-1 latency.Finding a cure for human immunodeficiency virus-1 infection.HIV latency is influenced by regions of the viral genome outside of the long terminal repeats and regulatory genes.LGIT In Vitro Latency Model in Primary and T Cell Lines to Test HIV-1 Reactivation Compounds.Establishment of HIV latency in primary CD4+ cells is due to epigenetic transcriptional silencing and P-TEFb restriction.Establishment and maintenance of HIV latency: model systems and opportunities for intervention.HIV latency in the humanized BLT mouse.The HIV-1 reservoir in eight patients on long-term suppressive antiretroviral therapy is stable with few genetic changes over timeSingle mutations in HIV integrase confer high-level resistance to raltegravir in primary human macrophages.
P2860
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P2860
Primary cell model for activation-inducible human immunodeficiency virus
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Primary cell model for activation-inducible human immunodeficiency virus
@en
Primary cell model for activation-inducible human immunodeficiency virus
@nl
type
label
Primary cell model for activation-inducible human immunodeficiency virus
@en
Primary cell model for activation-inducible human immunodeficiency virus
@nl
prefLabel
Primary cell model for activation-inducible human immunodeficiency virus
@en
Primary cell model for activation-inducible human immunodeficiency virus
@nl
P2093
P2860
P356
P1433
P1476
Primary cell model for activation-inducible human immunodeficiency virus
@en
P2093
Bryan Burke
Dimitrios N Vatakis
Gregory Bristol
Helen J Brown
Jerome A Zack
Matthew D Marsden
P2860
P304
P356
10.1128/JVI.02838-06
P407
P577
2007-05-02T00:00:00Z