Systemic endothelial activation occurs in both mild and severe malaria. Correlating dermal microvascular endothelial cell phenotype and soluble cell adhesion molecules with disease severity.
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Plasmodium falciparum histones induce endothelial proinflammatory response and barrier dysfunctionInhibition of endothelial activation: a new way to treat cerebral malaria?Fighting the Monster: Applying the Host Damage Framework to Human Central Nervous System InfectionsAbnormal PfEMP1/knob display on Plasmodium falciparum-infected erythrocytes containing hemoglobin variants: fresh insights into malaria pathogenesis and protectionCD19(+) B cells confer protection against experimental cerebral malaria in semi-immune rodent modelMicrofluidic modeling of cell-cell interactions in malaria pathogenesisCombinations of host biomarkers predict mortality among Ugandan children with severe malaria: a retrospective case-control studyInhaled nitric oxide reduces endothelial activation and parasite accumulation in the brain, and enhances survival in experimental cerebral malariaEndothelium-based biomarkers are associated with cerebral malaria in Malawian children: a retrospective case-control studyImpaired nitric oxide bioavailability and L-arginine reversible endothelial dysfunction in adults with falciparum malaria.Blood coagulation, inflammation, and malaria.Severe Plasmodium falciparum malaria is associated with circulating ultra-large von Willebrand multimers and ADAMTS13 inhibitionSerum angiopoietin-1 and -2 levels discriminate cerebral malaria from uncomplicated malaria and predict clinical outcome in African childrenA novel role for von Willebrand factor in the pathogenesis of experimental cerebral malariaWhole blood angiopoietin-1 and -2 levels discriminate cerebral and severe (non-cerebral) malaria from uncomplicated malaria.Plasmodium chabaudi-infected erythrocytes adhere to CD36 and bind to microvascular endothelial cells in an organ-specific waySingle molecule and multiple bond characterization of catch bond associated cytoadhesion in malaria.Does activation of the blood coagulation cascade have a role in malaria pathogenesis?Plasmodium falciparum-infected erythrocytes induce tissue factor expression in endothelial cells and support the assembly of multimolecular coagulation complexesIncreased levels of soluble Fas ligand in serum in Plasmodium falciparum malaria.Combined measurement of soluble and cellular ICAM-1 among children with Plasmodium falciparum malaria in Uganda.Cytoadherence phenotype of Plasmodium falciparum-infected erythrocytes is associated with specific pfemp-1 expression in parasites from children with cerebral malariaA potential role for plasma uric acid in the endothelial pathology of Plasmodium falciparum malaria.Malaria-induced splenic infarction.Tumor necrosis factor -α, interleukin-10, intercellular and vascular adhesion molecules are possible biomarkers of disease severity in complicated Plasmodium vivax isolates from Pakistan.Common and divergent immune response signaling pathways discovered in peripheral blood mononuclear cell gene expression patterns in presymptomatic and clinically apparent malaria.High plasma levels of soluble intercellular adhesion molecule (ICAM)-1 are associated with cerebral malaria.An analysis of the binding characteristics of a panel of recently selected ICAM-1 binding Plasmodium falciparum patient isolatesIncreased pulmonary pressures and myocardial wall stress in children with severe malariaMicrovascular obstruction and endothelial activation are independently associated with the clinical manifestations of severe falciparum malaria in adults: an observational study.Brain swelling and death in children with cerebral malariaInhibiting the Mammalian target of rapamycin blocks the development of experimental cerebral malaria.Angiopoietin-2 levels are associated with retinopathy and predict mortality in Malawian children with cerebral malaria: a retrospective case-control study*A quantitative analysis of the microvascular sequestration of malaria parasites in the human brain.Cytoadherence and virulence - the case of Plasmodium knowlesi malariaDoes Iron Increase the Risk of Malaria in Pregnancy?Cytoadherence and severe malaria.A clinicopathological correlation of the expression of the angiopoietin-Tie-2 receptor pathway in the brain of adults with Plasmodium falciparum malaria.C5 deficiency and C5a or C5aR blockade protects against cerebral malariaDiversity of the var gene family of Indonesian Plasmodium falciparum isolates.
P2860
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P2860
Systemic endothelial activation occurs in both mild and severe malaria. Correlating dermal microvascular endothelial cell phenotype and soluble cell adhesion molecules with disease severity.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Systemic endothelial activatio ...... lecules with disease severity.
@en
Systemic endothelial activatio ...... lecules with disease severity.
@nl
type
label
Systemic endothelial activatio ...... lecules with disease severity.
@en
Systemic endothelial activatio ...... lecules with disease severity.
@nl
prefLabel
Systemic endothelial activatio ...... lecules with disease severity.
@en
Systemic endothelial activatio ...... lecules with disease severity.
@nl
P2093
P2860
P1476
Systemic endothelial activatio ...... lecules with disease severity.
@en
P2093
A R Berendt
G D Turner
H P Nguyen
K C Gatter
K Louwrier
T H Nguyen
P2860
P304
P407
P577
1998-06-01T00:00:00Z