Neuropathic pain: early spontaneous afferent activity is the trigger.
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Chemokines and the pathophysiology of neuropathic painClinical significance of tumor necrosis factor-α inhibitors in the treatment of sciatica: a systematic review and meta-analysisPerioperative interventions to reduce chronic postsurgical painEnhanced excitability of primary sensory neurons and altered gene expression of neuronal ion channels in dorsal root ganglion in paclitaxel-induced peripheral neuropathyUrothelial Tight Junction Barrier Dysfunction Sensitizes Bladder AfferentsIntrathecal lidocaine pretreatment attenuates immediate neuropathic pain by modulating Nav1.3 expression and decreasing spinal microglial activationSensory stimulation prior to spinal cord injury induces post-injury dysesthesia in mice.Early afferent activity from the facet joint after painful trauma to its capsule potentiates neuronal excitability and glutamate signaling in the spinal cord.Blocking the mineralocorticoid receptor improves effectiveness of steroid treatment for low back pain in rats.Expression of inwardly rectifying potassium channels by an inducible adenoviral vector reduced the neuronal hyperexcitability and hyperalgesia produced by chronic compression of the spinal ganglion.Tetrodotoxin (TTX) as a therapeutic agent for pain.Pretreatment with intrathecal amitriptyline potentiates anti-hyperalgesic effects of post-injury intra-peritoneal amitriptyline following spinal nerve ligationImmature spinal cord neurons are dynamic regulators of adult nociceptive sensitivity.Multiple sites for generation of ectopic spontaneous activity in neurons of the chronically compressed dorsal root ganglion.Chronic spontaneous activity generated in the somata of primary nociceptors is associated with pain-related behavior after spinal cord injury.Perioperative nerve blockade: clues from the bench.Nerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.Highly localized interactions between sensory neurons and sprouting sympathetic fibers observed in a transgenic tyrosine hydroxylase reporter mouse.Local knockdown of the NaV1.6 sodium channel reduces pain behaviors, sensory neuron excitability, and sympathetic sprouting in rat models of neuropathic pain.Neuropathic pain as a process: reversal of chronification in an animal model.Pre-emptive treatment of lidocaine attenuates neuropathic pain and reduces pain-related biochemical markers in the rat cuneate nucleus in median nerve chronic constriction injury modelSympathetic sprouting near sensory neurons after nerve injury occurs preferentially on spontaneously active cells and is reduced by early nerve block.Dynamics of on-off neural firing patterns and stochastic effects near a sub-critical Hopf bifurcationBursting activity in myelinated sensory neurons plays a key role in pain behavior induced by localized inflammation of the rat sensory ganglionSynaptic structure and function in the mouse somatosensory cortex during chronic pain: in vivo two-photon imaging.Spontaneous Chronic Pain After Experimental Thoracotomy Revealed by Conditioned Place Preference: Morphine Differentiates Tactile Evoked Pain From Spontaneous PainSystemic antiinflammatory corticosteroid reduces mechanical pain behavior, sympathetic sprouting, and elevation of proinflammatory cytokines in a rat model of neuropathic painTopical Tetrodotoxin Attenuates Photophobia Induced by Corneal Injury in the Rat.Prolonged nerve blockade delays the onset of neuropathic painPeripheral nerve injury produces a sustained shift in the balance between glutamate release and uptake in the dorsal horn of the spinal cord.Targeting invading macrophage-derived PGE2, IL-6 and calcitonin gene-related peptide in injured nerve to treat neuropathic pain.Spinal neuronal plasticity is evident within 1 day after a painful cervical facet joint injury.Knockdown of sodium channel NaV1.6 blocks mechanical pain and abnormal bursting activity of afferent neurons in inflamed sensory ganglia.Upregulation of the sodium channel NaVβ4 subunit and its contributions to mechanical hypersensitivity and neuronal hyperexcitability in a rat model of radicular pain induced by local dorsal root ganglion inflammation.Development of a spontaneously active dorsal root ganglia assay using multiwell multielectrode arraysProtective effects of gabapentin on allodynia and alpha 2 delta 1-subunit of voltage-dependent calcium channel in spinal nerve-ligated ratsLocalized Sympathectomy Reduces Mechanical Hypersensitivity by Restoring Normal Immune Homeostasis in Rat Models of Inflammatory Pain.Cytokine and chemokine regulation of sensory neuron function.Behavioral models of pain states evoked by physical injury to the peripheral nerve.Large A-fiber activity is required for microglial proliferation and p38 MAPK activation in the spinal cord: different effects of resiniferatoxin and bupivacaine on spinal microglial changes after spared nerve injury.
P2860
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P2860
Neuropathic pain: early spontaneous afferent activity is the trigger.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Neuropathic pain: early spontaneous afferent activity is the trigger.
@en
Neuropathic pain: early spontaneous afferent activity is the trigger.
@nl
type
label
Neuropathic pain: early spontaneous afferent activity is the trigger.
@en
Neuropathic pain: early spontaneous afferent activity is the trigger.
@nl
prefLabel
Neuropathic pain: early spontaneous afferent activity is the trigger.
@en
Neuropathic pain: early spontaneous afferent activity is the trigger.
@nl
P2093
P2860
P1433
P1476
Neuropathic pain: early spontaneous afferent activity is the trigger.
@en
P2093
Johanna T A Meij
Judith A Strong
Jun-Ming Zhang
Wenrui Xie
P2860
P304
P356
10.1016/J.PAIN.2005.04.017
P407
P577
2005-08-01T00:00:00Z