Long-term potentiation can be induced in the CA1 region of hippocampus in the absence of αCaMKII T286-autophosphorylation.
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CX3CL1/CX3CR1 in Alzheimer's Disease: A Target for NeuroprotectionCaMKII Autophosphorylation Is Necessary for Optimal Integration of Ca2+ Signals during LTP Induction, but Not Maintenance.Memory Erasure Experiments Indicate a Critical Role of CaMKII in Memory Storage.CaMKII mediates stimulus specificity in early odor preference learning in rats.Impaired Hippocampal Synaptic Plasticity and Enhanced Excitatory Transmission in a Novel Animal Model of Autism Spectrum Disorders with Telomerase Reverse Transcriptase Overexpression.
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Long-term potentiation can be induced in the CA1 region of hippocampus in the absence of αCaMKII T286-autophosphorylation.
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2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
@zh-tw
2014年论文
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2014年论文
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2014年论文
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name
Long-term potentiation can be ...... MKII T286-autophosphorylation.
@en
Long-term potentiation can be ...... MKII T286-autophosphorylation.
@nl
type
label
Long-term potentiation can be ...... MKII T286-autophosphorylation.
@en
Long-term potentiation can be ...... MKII T286-autophosphorylation.
@nl
prefLabel
Long-term potentiation can be ...... MKII T286-autophosphorylation.
@en
Long-term potentiation can be ...... MKII T286-autophosphorylation.
@nl
P2860
P356
P1433
P1476
Long-term potentiation can be ...... aMKII T286-autophosphorylation
@en
P2093
Karl Peter Giese
Laurence Ris
P2860
P304
P356
10.1101/LM.035972.114
P577
2014-10-16T00:00:00Z