Chronic methylphenidate treatment enhances striatal dopamine neurotransmission after experimental traumatic brain injury. - Wikidata - wikimedia - TriplyDB

Chronic methylphenidate treatment enhances striatal dopamine neurotransmission after experimental traumatic brain injury.

Chronic methylphenidate treatment enhances striatal dopamine neurotransmission after experimental traumatic brain injury.

http://www.wikidata.org/entity/Q42128651

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Catecholamines and cognition after traumatic brain injury Developing a clinically relevant model of cognitive training after experimental traumatic brain injury.Amantadine ameliorates dopamine-releasing deficits and behavioral deficits in rats after fluid percussion injury The Controlled Cortical Impact Model: Applications, Considerations for Researchers, and Future Directions Emerging therapies in traumatic brain injury Found in translation: Understanding the biology and behavior of experimental traumatic brain injury Background Signal as an in Situ Predictor of Dopamine Oxidation Potential: Improving Interpretation of Fast-Scan Cyclic Voltammetry Data.Old dog, new tricks: the attentional set-shifting test as a novel cognitive behavioral task after controlled cortical impact injury.Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis Methamphetamine-induced neurotoxicity disrupts pharmacologically evoked dopamine transients in the dorsomedial and dorsolateral striatum A Rehabilomics framework for personalized and translational rehabilitation research and care for individuals with disabilities: Perspectives and considerations for spinal cord injury.Experimental traumatic brain injury alters ethanol consumption and sensitivity.Biologic and plastic effects of experimental traumatic brain injury treatment paradigms and their relevance to clinical rehabilitation.Targeting Dopamine in Acute Traumatic Brain Injury Visual Priming Enhances the Effects of Nonspatial Cognitive Rehabilitation Training on Spatial Learning After Experimental Traumatic Brain Injury.Mental Health Implications of Traumatic Brain Injury (TBI) in Children and Youth.Posttraumatic Brain Injury Cognitive Performance Is Moderated by Variation Within ANKK1 and DRD2 Genes.Modeling Fast-scan Cyclic Voltammetry Data from Electrically Stimulated Dopamine Neurotransmission Data Using QNsim1.0.Traumatic Brain Injury Impairs Soluble N-Ethylmaleimide-Sensitive Factor Attachment Protein Receptor Complex Formation and Alters Synaptic Vesicle Distribution in the Hippocampus.Catecholaminergic based therapies for functional recovery after TBI Pharmacotherapy of traumatic brain injury: state of the science and the road forward: report of the Department of Defense Neurotrauma Pharmacology Workgroup.Ventricular fibrillation cardiac arrest produces a chronic striatal hyperdopaminergic state that is worsened by methylphenidate treatment.The influence of genetic variants on striatal dopamine transporter and D2 receptor binding after TBI.A study on the mechanism by which MDMA protects against dopaminergic dysfunction after minimal traumatic brain injury (mTBI) in mice.Differential expression of microRNAs in the brains of mice subjected to increasing grade of mild traumatic brain injury.The Therapeutic Efficacy of Environmental Enrichment and Methylphenidate Alone and in Combination after Controlled Cortical Impact Injury.Alpha-synuclein (SNCA) polymorphisms exert protective effects on memory after mild traumatic brain injury.Traumatic Brain Injuries during Development: Implications for Alcohol Abuse.Controlled cortical impact injury influences methylphenidate-induced changes in striatal dopamine neurotransmission.COMT and ANKK1 Genetics Interact With Depression to Influence Behavior Following Severe TBI: An Initial Assessment Lateral Fluid Percussion Injury Impairs Hippocampal Synaptic Soluble N-Ethylmaleimide Sensitive Factor Attachment Protein Receptor Complex Formation.The dopamine and cAMP regulated phosphoprotein, 32 kDa (DARPP-32) signaling pathway: a novel therapeutic target in traumatic brain injury.Fast-scan cyclic voltammetry demonstrates that L-DOPA produces dose-dependent regionally selective, bimodal effects on striatal dopamine kinetics in vivo.Impact of Traumatic Brain Injury on Dopaminergic Transmission.The intriguing effects of ecstasy (MDMA) on cognitive function in mice subjected to a minimal traumatic brain injury (mTBI).Executive (dys)function after traumatic brain injury: special considerations for behavioral pharmacology

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P2860

Chronic methylphenidate treatment enhances striatal dopamine neurotransmission after experimental traumatic brain injury.

http://www.wikidata.org/entity/Q42128651

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Chronic methylphenidate treatm ...... mental traumatic brain injury.

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Chronic methylphenidate treatm ...... mental traumatic brain injury.

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Chronic methylphenidate treatm ...... mental traumatic brain injury.

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Chronic methylphenidate treatm ...... mental traumatic brain injury.

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J.1471-4159.2008.05840.X

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Journal of Neurochemistry

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Chronic methylphenidate treatm ...... imental traumatic brain injury

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Adrian C Michael

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Amina S Khan

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Amy K Wagner

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C Edward Dixon

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F Ryan Santos

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Gonzalo E Torres

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Laura L Drewencki

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Xiangbai Chen

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P2860

Substrates regulate gamma-aminobutyric acid transporters in a syntaxin 1A-dependent manner.

Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum

Profound neuronal plasticity in response to inactivation of the dopamine transporter

The up-regulation of the striatal dopamine transporter's activity by cAMP is PKA-, CaMK II- and phosphatase-dependent

Hyperactivity and impaired response habituation in hyperdopaminergic mice.

Dopamine D(2) receptors regulate tyrosine hydroxylase activity and phosphorylation at Ser40 in rat striatum.

Glutamate regulates the spontaneous and evoked release of dopamine in the rat striatum.

Regulation of immediate early gene expression and AP-1 binding in the rat nucleus accumbens by chronic cocaine.

Electrochemical monitoring of biogenic amine neurotransmission in real time.

Patterns of gene expression and behavior induced by chronic dopamine treatments.

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986-997

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10.1111/J.1471-4159.2008.05840.X

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19077052

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corpus striatum

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2692956

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