Overexpression of mitochondrial Hsp70/Hsp75 in rat brain protects mitochondria, reduces oxidative stress, and protects from focal ischemia.
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Advances in astrocyte-targeted approaches for stroke therapy: an emerging role for mitochondria and microRNASInflammation, mitochondria, and the inhibition of adult neurogenesisHuman mitochondrial Hsp70 (mortalin): shedding light on ATPase activity, interaction with adenosine nucleotides, solution structure and domain organizationComparison of brain transcriptome of the greater horseshoe bats (Rhinolophus ferrumequinum) in active and torpid episodesHeat shock proteins: cellular and molecular mechanisms in the central nervous system.Gene therapy with mitochondrial heat shock protein 70 suppresses visual loss and optic atrophy in experimental autoimmune encephalomyelitis.Mitochondrial protection attenuates inflammation-induced impairment of neurogenesis in vitro and in vivo.TRAP1 controls mitochondrial fusion/fission balance through Drp1 and Mff expressionAstrocyte targeted overexpression of Hsp72 or SOD2 reduces neuronal vulnerability to forebrain ischemia.Increased brain injury and worsened neurological outcome in interleukin-4 knockout mice after transient focal cerebral ischemiaMicroRNA-200c contributes to injury from transient focal cerebral ischemia by targeting ReelinEffect of polyphenols on oxidative stress and mitochondrial dysfunction in neuronal death and brain edema in cerebral ischemia.C-terminal heat shock protein 90 inhibitor decreases hyperglycemia-induced oxidative stress and improves mitochondrial bioenergetics in sensory neurons.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Diabetic peripheral neuropathy: should a chaperone accompany our therapeutic approach?Effects of heat shock protein 72 (Hsp72) on evolution of astrocyte activation following stroke in the mouse.Aberrantly upregulated TRAP1 is required for tumorigenesis of breast cancer.microRNAs: innovative targets for cerebral ischemia and stroke.Aqueous extract from the Withania somnifera leaves as a potential anti-neuroinflammatory agent: a mechanistic study.Heat shock protein 70 protects PC12 cells against ischemia-hypoxia/reoxygenation by maintaining intracellular Ca(2+) homeostasis.Molecular interactions of Bcl-2 and Bcl-xL with mortalin: identification and functional characterizationMicroRNAs regulate the chaperone network in cerebral ischemia.Compartmentalized cancer drug discovery targeting mitochondrial Hsp90 chaperones.Oxidative protein folding in the endoplasmic reticulum: tight links to the mitochondria-associated membrane (MAM).Measuring endogenous 5-HT release by emission tomography: promises and pitfalls.Astrocytes: targets for neuroprotection in stroke.Mitochondrial compartmentalized protein folding and tumor cell survival.New insights into TRAP1 pathway.ER-Mitochondria Crosstalk during Cerebral Ischemia: Molecular Chaperones and ER-Mitochondrial Calcium Transfer.The use of microRNAs to modulate redox and immune response to stroke.Regulators of mitochondrial Ca(2+) homeostasis in cerebral ischemia.Mortalin, apoptosis, and neurodegeneration.The mitochondrial permeability transition pore and its adaptive responses in tumor cells.Tumor Necrosis Factor-Associated Protein 1 (TRAP1) is Released from the Mitochondria Following 6-hydroxydopamine Treatment.A Novel In Vitro CypD-Mediated p53 Aggregation Assay Suggests a Model for Mitochondrial Permeability Transition by Chaperone Systems.Inhibition of succinate dehydrogenase by the mitochondrial chaperone TRAP1 has anti-oxidant and anti-apoptotic effects on tumor cells.SHetA2 interference with mortalin binding to p66shc and p53 identified using drug-conjugated magnetic microspheresThe Chaperone TRAP1 As a Modulator of the Mitochondrial Adaptations in Cancer Cells.In vivo imaging of induction of heat-shock protein-70 gene expression with fluorescence reflectance imaging and intravital confocal microscopy following brain ischaemia in reporter mice.Impairment of oxidative stress-induced heme oxygenase-1 expression by the defect of Parkinson-related gene of PINK1.
P2860
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P2860
Overexpression of mitochondrial Hsp70/Hsp75 in rat brain protects mitochondria, reduces oxidative stress, and protects from focal ischemia.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Overexpression of mitochondria ...... protects from focal ischemia.
@en
Overexpression of mitochondria ...... protects from focal ischemia.
@nl
type
label
Overexpression of mitochondria ...... protects from focal ischemia.
@en
Overexpression of mitochondria ...... protects from focal ischemia.
@nl
prefLabel
Overexpression of mitochondria ...... protects from focal ischemia.
@en
Overexpression of mitochondria ...... protects from focal ischemia.
@nl
P2093
P2860
P356
P1476
Overexpression of mitochondria ...... protects from focal ischemia.
@en
P2093
John F Emery
Ludmila A Voloboueva
Rona G Giffard
YiBing Ouyang
P2860
P304
P356
10.1038/JCBFM.2008.125
P577
2008-11-05T00:00:00Z