A novel retinoic acid-response element requires an enhancer element mediator for transcriptional activation.

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Enhancing enhancers: new complexities in the retinoid regulation of gene expression.

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Q42808380-77CDCB2C-7C3A-4BB2-AEAE-E097D33E78C0

P2860

A novel retinoic acid-response element requires an enhancer element mediator for transcriptional activation.

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http://www.wikidata.org/entity/Q42176427

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A novel retinoic acid-response ...... or transcriptional activation.

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A novel retinoic acid-response ...... or transcriptional activation.

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Item

label

A novel retinoic acid-response ...... or transcriptional activation.

@en

A novel retinoic acid-response ...... or transcriptional activation.

@nl

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A novel retinoic acid-response ...... or transcriptional activation.

@en

A novel retinoic acid-response ...... or transcriptional activation.

@nl

P1476

A novel retinoic acid-response ...... for transcriptional activation

@en

P2093

Gwen C Miller

http://www.w3.org/2001/XMLSchema#string

James W Clarke

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Jennifer L Potter

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Laura R Harris

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Minna Sevakivi

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Olli-Pekka Kamarainen

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P2860

Splicing mutations of 54-bp exons in the COL11A1 gene cause Marshall syndrome, but other mutations cause overlapping Marshall/Stickler phenotypes

Stickler syndrome: further mutations in COL11A1 and evidence for additional locus heterogeneity

Autosomal recessive disorder otospondylomegaepiphyseal dysplasia is associated with loss-of-function mutations in the COL11A2 gene.

Oto- spondylo-megaepiphyseal dysplasia (OSMED): clinical description of three patients homozygous for a missense mutation in the COL11A2 gene

The human alpha 2(XI) collagen gene (COL11A2): completion of coding information, identification of the promoter sequence, and precise localization within the major histocompatibility complex reveal overlap with the KE5 gene

Marshall syndrome associated with a splicing defect at the COL11A1 locus.

Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

Widely distributed mutations in the COL2A1 gene produce achondrogenesis type II/hypochondrogenesis

Nuclear hormone receptors and gene expression

Stickler syndrome without eye involvement is caused by mutations in COL11A2, the gene encoding the alpha2(XI) chain of type XI collagen

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37-43

http://www.w3.org/2001/XMLSchema#string

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scholarly article

P356

10.1042/BJ20040715

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English

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Pt 1

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P478

383

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P50

Laura C Bridgewater

P577

2004-10-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

8501042

http://www.w3.org/2001/XMLSchema#string

P698

15206905

http://www.w3.org/2001/XMLSchema#string

P932

1134041

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A novel retinoic acid-response element requires an enhancer element mediator for transcriptional activation.

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P2860

P2860

A novel retinoic acid-response element requires an enhancer element mediator for transcriptional activation.

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P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P5875

P698

P932