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HIV/AIDS. Persistence by proliferation?

HIV/AIDS. Persistence by proliferation?

http://www.wikidata.org/entity/Q42206888

about

Bioinformatics and HIV latency.Residual Viremia in Treated HIV+ Individuals Eradicating HIV-1 infection: seeking to clear a persistent pathogen.HIV Reactivation from Latency after Treatment Interruption Occurs on Average Every 5-8 Days--Implications for HIV Remission.Host Factors in Retroviral Integration and the Selection of Integration Target Sites.Position-effect variegation revisited: HUSHing up heterochromatin in human cells.

P2860

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P2860

HIV/AIDS. Persistence by proliferation?

http://www.wikidata.org/entity/Q42206888

description

2014 nî lūn-bûn

@nan

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

2014年论文

@zh

2014年论文

@zh-cn

name

HIV/AIDS. Persistence by proliferation?

@en

HIV/AIDS. Persistence by proliferation?

@nl

type

label

HIV/AIDS. Persistence by proliferation?

@en

HIV/AIDS. Persistence by proliferation?

@nl

prefLabel

HIV/AIDS. Persistence by proliferation?

@en

HIV/AIDS. Persistence by proliferation?

@nl

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SCIENCE.1257426

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P1476

HIV/AIDS. Persistence by proliferation?

@en

P2093

Frederic Bushman

http://www.w3.org/2001/XMLSchema#string

P2860

Residual human immunodeficiency virus type 1 viremia in some patients on antiretroviral therapy is dominated by a small number of invariant clones rarely found in circulating CD4+ T cells

Transfusion independence and HMGA2 activation after gene therapy of human β-thalassaemia

Identification of a reservoir for HIV-1 in patients on highly active antiretroviral therapy

Presence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapy

Recovery of replication-competent HIV despite prolonged suppression of plasma viremia

HIV-1 integration in the human genome favors active genes and local hotspots

HIV reservoir size and persistence are driven by T cell survival and homeostatic proliferation

Recurrent HIV-1 integration at the BACH2 locus in resting CD4+ T cell populations during effective highly active antiretroviral therapy.

HIV latency. Specific HIV integration sites are linked to clonal expansion and persistence of infected cells

Clonal sequences recovered from plasma from patients with residual HIV-1 viremia and on intensified antiretroviral therapy are identical to replicating viral RNAs recovered from circulating resting CD4+ T cells.

P304

143-144

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P31

scholarly article

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10.1126/SCIENCE.1257426

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P433

6193

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345

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David M. Margolis

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2014-07-01T00:00:00Z

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P5875

264642379

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25013050

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