MBD5 regulates iron metabolism via methylation-independent genomic targeting of Fth1 through KAT2A in mice.
about
Disruption of Mbd5 in mice causes neuronal functional deficits and neurobehavioral abnormalities consistent with 2q23.1 microdeletion syndrome.The methyl binding domain containing protein MBD5 is a transcriptional regulator responsible for 2q23.1 deletion syndrome.Clinical and Molecular Aspects of MBD5-Associated Neurodevelopmental Disorder (MAND).Effects of cigarette smoke, cessation and switching to a candidate modified risk tobacco product on the liver in Apoe -/- mice--a systems toxicology analysis.Transcriptional Profiling of Dibenzo[def,p]chrysene-induced Spleen Atrophy Provides Mechanistic Insights into its Immunotoxicity in MutaMouse.Methotrexate combined with methylprednisolone for the recovery of motor function and differential gene expression in rats with spinal cord injury.Cystathionine β-synthase is required for body iron homeostasis.
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P2860
MBD5 regulates iron metabolism via methylation-independent genomic targeting of Fth1 through KAT2A in mice.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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2014年论文
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name
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice.
@en
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice.
@nl
type
label
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice.
@en
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice.
@nl
prefLabel
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice.
@en
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice.
@nl
P2860
P50
P356
P1476
MBD5 regulates iron metabolism ...... of Fth1 through KAT2A in mice
@en
P2093
Yuanyuan Shen
P2860
P304
P356
10.1111/BJH.12863
P407
P577
2014-04-18T00:00:00Z