HIV-1 protein Tat produces biphasic changes in NMDA-evoked increases in intracellular Ca2+ concentration via activation of Src kinase and nitric oxide signaling pathways.
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The cross-talk of HIV-1 Tat and methamphetamine in HIV-associated neurocognitive disordersMechanisms of HIV-1 Tat neurotoxicity via CDK5 translocation and hyper-activation: role in HIV-associated neurocognitive disordersHIV-1 Tat-induced changes in synaptically-driven network activity adapt during prolonged exposure.Opinion: Inhibition of Blood-Brain Barrier Repair as a Mechanism in HIV-1 Disease.HIV-1 Tat activates a RhoA signaling pathway to reduce NMDA-evoked calcium responses in hippocampal neurons via an actin-dependent mechanismMSM ameliorates HIV-1 Tat induced neuronal oxidative stress via rebalance of the glutathione cycleHIV-1 subtypes B and C Tat differentially impact synaptic plasticity expression and implicates HIV-associated neurocognitive disorders.HIV-1 gp120Bal down-Regulates Phosphorylated NMDA Receptor Subunit 1 in Cortical Neurons via Activation of Glutamate and Chemokine Receptors.Interleukin-1β activates an Src family kinase to stimulate the plasma membrane Ca2+ pump in hippocampal neurons.NMDARs Adapt to Neurotoxic HIV Protein Tat Downstream of a GluN2A-Ubiquitin Ligase Signaling Pathway.Synaptic signalling and its interface with neuropathologies: snapshots from the past, present and future.Genome-wide association mapping and pathway analysis of leukosis incidence in a US Holstein cattle population.Human Immunodeficiency Virus Promotes Mitochondrial Toxicity.A GluN2B-Selective NMDAR Antagonist Reverses Synapse Loss and Cognitive Impairment Produced by the HIV-1 Protein Tat.Dynamic plasticity of NMDA receptor-mediated calcium entry in neurons exposed to HIV-tat.Monoacylglycerol lipase inhibitor JZL184 prevents HIV-1 gp120-induced synapse loss by altering endocannabinoid signaling.Astrocytic metabolic switch is a novel etiology for Cocaine and HIV-1 Tat-mediated neurotoxicity.HIV-1 Tat alters neuronal intrinsic excitability.Methamphetamine augment HIV-1 Tat mediated memory deficits by altering the expression of synaptic proteins and neurotrophic factors.
P2860
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P2860
HIV-1 protein Tat produces biphasic changes in NMDA-evoked increases in intracellular Ca2+ concentration via activation of Src kinase and nitric oxide signaling pathways.
description
2014 nî lūn-bûn
@nan
2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
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2014年论文
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2014年论文
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2014年论文
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name
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@en
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@nl
type
label
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@en
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@nl
prefLabel
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@en
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@nl
P2093
P2860
P356
P1476
HIV-1 protein Tat produces bip ...... tric oxide signaling pathways.
@en
P2093
Kelly A Krogh
Nicole Wydeven
Stanley A Thayer
P2860
P304
P356
10.1111/JNC.12724
P407
P577
2014-04-19T00:00:00Z