The origin of aging: imperfectness-driven non-random damage defines the aging process and control of lifespan.
about
Aging: progressive decline in fitness due to the rising deleteriome adjusted by genetic, environmental, and stochastic processesWhich Is the Most Significant Cause of Aging?Discovery of plant extracts that greatly delay yeast chronological aging and have different effects on longevity-defining cellular processesAging is not a disease: implications for intervention.Innovating aging: promises and pitfalls on the road to life extension.Increased DNA methylation in the suicide brain.Stability analysis of a model gene network links aging, stress resistance, and negligible senescence.Theories of aging: an ever-evolving fieldGenomes of Strongylocentrotus franciscanus and Lytechinus variegatus: are there any genomic explanations for the two order of magnitude difference in the lifespan of sea urchins?The temporal scaling of Caenorhabditis elegans ageing.Redox Pioneer: Professor Vadim N. Gladyshev.Analysis of cancer genomes reveals basic features of human aging and its role in cancer development.Age-associated molecular changes are deleterious and may modulate life span through diet.Age- and diet-associated metabolome remodeling characterizes the aging process driven by damage accumulation.Directly Reprogrammed Human Neurons Retain Aging-Associated Transcriptomic Signatures and Reveal Age-Related Nucleocytoplasmic Defects.The aging-disease false dichotomy: understanding senescence as pathology.Convergent adaptation of cellular machineries in the evolution of large body masses and long life spans.Using DNA Methylation Profiling to Evaluate Biological Age and Longevity Interventions.Evidence that mutation accumulation does not cause aging in Saccharomyces cerevisiae.Suppression of transcriptional drift extends C. elegans lifespan by postponing the onset of mortality.The free radical theory of aging is dead. Long live the damage theory!Caloric restriction extends yeast chronological lifespan via a mechanism linking cellular aging to cell cycle regulation, maintenance of a quiescent state, entry into a non-quiescent state and survival in the non-quiescent state.The role of glycation in the pathogenesis of aging and its prevention through herbal products and physical exercise.Pathways to Brain Aging and Their Modifiers: Free-Radical-Induced Energetic and Neural Decline in Senescence (FRIENDS) Model - A Mini-Review.
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P2860
The origin of aging: imperfectness-driven non-random damage defines the aging process and control of lifespan.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
The origin of aging: imperfect ...... ocess and control of lifespan.
@en
The origin of aging: imperfect ...... ocess and control of lifespan.
@nl
type
label
The origin of aging: imperfect ...... ocess and control of lifespan.
@en
The origin of aging: imperfect ...... ocess and control of lifespan.
@nl
prefLabel
The origin of aging: imperfect ...... ocess and control of lifespan.
@en
The origin of aging: imperfect ...... ocess and control of lifespan.
@nl
P2860
P1433
P1476
The origin of aging: imperfect ...... ocess and control of lifespan.
@en
P2093
Vadim N Gladyshev
P2860
P304
P356
10.1016/J.TIG.2013.05.004
P577
2013-06-13T00:00:00Z