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Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

http://www.wikidata.org/entity/Q42274604

about

A mitochondria-targeted inhibitor of cytochrome c peroxidase mitigates radiation-induced death Effects of ionizing radiation on biological molecules--mechanisms of damage and emerging methods of detection Sirt3, mitochondrial ROS, ageing, and carcinogenesis.Hydrogen peroxide fuels aging, inflammation, cancer metabolism and metastasis: the seed and soil also needs "fertilizer".Sirt3-mediated deacetylation of evolutionarily conserved lysine 122 regulates MnSOD activity in response to stress.Reactive oxygen species mediate microRNA-302 regulation of AT-rich interacting domain 4a and C-C motif ligand 5 expression during transitions between quiescence and proliferation Inhibition of p38 MAPK sensitizes tumour cells to cisplatin-induced apoptosis mediated by reactive oxygen species and JNK.Redox-modulated phenomena and radiation therapy: the central role of superoxide dismutases.Ionizing radiation-induced metabolic oxidative stress and prolonged cell injury.RAC2-P38 MAPK-dependent NADPH oxidase activity is associated with the resistance of quiescent cells to ionizing radiation.Forkhead box M1 regulates quiescence-associated radioresistance of human head and neck squamous carcinoma cells Mitochondrial fission mediates ceramide-induced metabolic disruption in skeletal muscle.Radiation interactions with biological systems.Disruption of Sag/Rbx2/Roc2 induces radiosensitization by increasing ROS levels and blocking NF-kappaB activation in mouse embryonic stem cells.Preventing Dr. Jekyll from becoming Mr. Hyde: is manganese superoxide dismutase the key to prevent radiation-induced neoplastic transformation?Near-infrared exposure changes cellular responses to ionizing radiation.Selenoprotein P inhibits radiation-induced late reactive oxygen species accumulation and normal cell injury.Redox Paradox: A novel approach to therapeutics-resistant cancer.

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P2860

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

http://www.wikidata.org/entity/Q42274604

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年论文

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name

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

@en

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

@nl

type

label

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

@en

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

@nl

prefLabel

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

@en

Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

@nl

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Mitochondrial ROS and radiation induced transformation in mouse embryonic fibroblasts.

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Amanda L Kalen

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Changbin Du

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Douglas R Spitz

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Joseph J Cullen

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Larry W Oberley

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Leena Chaudhuri

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Prabhat C Goswami

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Venkatasubbaiah A Venkatesha

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Zhen Gao

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P2860

DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation

The multiple checkpoint functions of CHK1 and CHK2 in maintenance of genome stability

Establishment and characterization of a cloned line of C3H mouse embryo cells sensitive to postconfluence inhibition of division.

Manganese superoxide dismutase-mediated gene expression in radiation-induced adaptive responses

Metabolic oxidation/reduction reactions and cellular responses to ionizing radiation: a unifying concept in stress response biology.

Chronic oxidative stress and radiation-induced late normal tissue injury: a review.

Mechanism of the tumor suppressive effect of MnSOD overexpression.

Catalase ameliorates polychlorinated biphenyl-induced cytotoxicity in nonmalignant human breast epithelial cells

Role of superoxide dismutase in cancer: a review.

Enzymatic activity is necessary for the tumor-suppressive effects of MnSOD.

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