Peptide-induced infant status epilepticus causes neuronal death and synaptic reorganization.
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Prenatal programming of human neurological functionNeurobehavioral risk is associated with gestational exposure to stress hormones.Hippocampal neuroplasticity induced by early-life stress: functional and molecular aspects.Abnormal dendritic maturation of developing cortical neurons exposed to corticotropin releasing hormone (CRH): Insights into effects of prenatal adversity?Correlated memory defects and hippocampal dendritic spine loss after acute stress involve corticotropin-releasing hormone signaling.Mossy fiber plasticity and enhanced hippocampal excitability, without hippocampal cell loss or altered neurogenesis, in an animal model of prolonged febrile seizuresCellular and molecular mechanisms of hippocampal activation by acute stress are age-dependent.Corticotropin-releasing hormone (CRH) downregulates the function of its receptor (CRF1) and induces CRF1 expression in hippocampal and cortical regions of the immature rat brain.How do the many etiologies of West syndrome lead to excitability and seizures? The corticotropin releasing hormone excess hypothesis.Do seizures affect the developing brain? Lessons from the laboratory.Stress and the developing hippocampus: a double-edged sword?Immunocytochemical distribution of corticotropin-releasing hormone receptor type-1 (CRF(1))-like immunoreactivity in the mouse brain: light microscopy analysis using an antibody directed against the C-terminus.Neuronal activity and stress differentially regulate hippocampal and hypothalamic corticotropin-releasing hormone expression in the immature ratThe in vivo proconvulsant effects of corticotropin releasing hormone in the developing rat are independent of ionotropic glutamate receptor activationInfantile spasms: hypothesis-driven therapy and pilot human infant experiments using corticotropin-releasing hormone receptor antagonists.Neuropeptide-mediated excitability: a key triggering mechanism for seizure generation in the developing brainThe pro-convulsant actions of corticotropin-releasing hormone in the hippocampus of infant ratsSeizure-induced neuronal injury: vulnerability to febrile seizures in an immature rat modelThe developmental profile of the corticotropin releasing factor receptor (CRF2) in rat brain predicts distinct age-specific functions.The CRF1 receptor mediates the excitatory actions of corticotropin releasing factor (CRF) in the developing rat brain: in vivo evidence using a novel, selective, non-peptide CRF receptor antagonistThe effect of 'Astressin', a novel antagonist of corticotropin releasing hormone (CRH), on CRH-induced seizures in the infant rat: comparison with two other antagonists.Selective death of hippocampal CA3 pyramidal cells with mossy fiber afferents after CRH-induced status epilepticus in infant ratsEpileptogenesis in the developing brain: what can we learn from animal models?Long-term, progressive hippocampal cell loss and dysfunction induced by early-life administration of corticotropin-releasing hormone reproduce the effects of early-life stress.Exposure to prenatal psychobiological stress exerts programming influences on the mother and her fetus.Maternal corticotropin-releasing hormone and habituation in the human fetus.Corticotropin-releasing hormone (CRH)-containing neurons in the immature rat hippocampal formation: light and electron microscopic features and colocalization with glutamate decarboxylase and parvalbumin.Corticotropin releasing hormone antagonist does not prevent adrenalectomy-induced apoptosis in the dentate gyrus of the rat hippocampus.Long-term neuroplasticity and functional consequences of single versus recurrent early-life seizures.Epileptogenesis during development: injury, circuit recruitment, and plasticity.Systemic adrenocorticotropic hormone administration down-regulates the expression of corticotropin-releasing hormone (CRH) and CRH-binding protein in infant rat hippocampus.Mossy fiber sprouting after recurrent seizures during early development in rats.Elevated corticotropin releasing hormone/corticotropin releasing hormone-R1 expression in postmortem brain obtained from children with generalized epilepsy.
P2860
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P2860
Peptide-induced infant status epilepticus causes neuronal death and synaptic reorganization.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Peptide-induced infant status ...... h and synaptic reorganization.
@en
Peptide-induced infant status ...... h and synaptic reorganization.
@nl
type
label
Peptide-induced infant status ...... h and synaptic reorganization.
@en
Peptide-induced infant status ...... h and synaptic reorganization.
@nl
prefLabel
Peptide-induced infant status ...... h and synaptic reorganization.
@en
Peptide-induced infant status ...... h and synaptic reorganization.
@nl
P2860
P1433
P1476
Peptide-induced infant status ...... h and synaptic reorganization.
@en
P2093
P2860
P304
P356
10.1097/00001756-199501000-00013
P577
1995-01-01T00:00:00Z