The role of the epithelial Na(+) channel (ENaC) in high AVP but low aldosterone states.
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Inhibition of ENaC by endothelin-1Activation of ENaC by AVP contributes to the urinary concentrating mechanism and dilution of plasma.Cyclic-AMP regulates postnatal development of neural and behavioral responses to NaCl in rats.Renal vasoconstriction by vasopressin V1a receptors is modulated by nitric oxide, prostanoids, and superoxide but not the ADP ribosyl cyclase CD38.
P2860
The role of the epithelial Na(+) channel (ENaC) in high AVP but low aldosterone states.
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2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
The role of the epithelial Na
@nl
The role of the epithelial Na(+) channel (ENaC) in high AVP but low aldosterone states.
@en
type
label
The role of the epithelial Na
@nl
The role of the epithelial Na(+) channel (ENaC) in high AVP but low aldosterone states.
@en
prefLabel
The role of the epithelial Na
@nl
The role of the epithelial Na(+) channel (ENaC) in high AVP but low aldosterone states.
@en
P2860
P356
P1476
The role of the epithelial Na(+) channel (ENaC) in high AVP but low aldosterone states
@en
P2093
James D Stockand
P2860
P356
10.3389/FPHYS.2012.00304
P577
2012-07-31T00:00:00Z