Ca2+ activated Cl- channels as targets for analgesics.

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Ca2+ activated Cl- channels as targets for analgesics.

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http://www.wikidata.org/entity/Q42341759

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2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

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2017年论文

@zh

2017年论文

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Ca2+ activated Cl- channels as targets for analgesics.

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Ca2+ activated Cl- channels as targets for analgesics.

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Ca2+ activated Cl- channels as targets for analgesics.

@en

Ca2+ activated Cl- channels as targets for analgesics.

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Ca2+ activated Cl- channels as targets for analgesics.

@en

Ca2+ activated Cl- channels as targets for analgesics.

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P1476

Ca2+ activated Cl- channels as targets for analgesics

@en

P2093

Isabella Salzer

http://www.w3.org/2001/XMLSchema#string

Klaus Schicker

http://www.w3.org/2001/XMLSchema#string

P2860

Cellular functions of TMEM16/anoctamin

Ani9, A Novel Potent Small-Molecule ANO1 Inhibitor with Negligible Effect on ANO2

The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl- channels.

Anoctamin-1 Cl(-) channels in nociception: activation by an N-aroylaminothiazole and capsaicin and inhibition by T16A[inh]-A01

Pain-enhancing mechanism through interaction between TRPV1 and anoctamin 1 in sensory neurons.

Anoctamin 1 mediates thermal pain as a heat sensor.

Anoctamin 1 contributes to inflammatory and nerve-injury induced hypersensitivity.

Control of sensory neuron excitability by serotonin involves 5HT2C receptors and Ca(2+)-activated chloride channels.

P304

45038-45039

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P31

scholarly article

editorial

P356

10.18632/ONCOTARGET.18354

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Stefan Boehm

Klaus Schicker

Isabella Salzer

P577

2017-07-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28586764

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P932

5542162

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