Gut-derived lipopolysaccharide augments adipose macrophage accumulation but is not essential for impaired glucose or insulin tolerance in mice. - Wikidata - wikimedia - TriplyDB

Gut-derived lipopolysaccharide augments adipose macrophage accumulation but is not essential for impaired glucose or insulin tolerance in mice.

Gut-derived lipopolysaccharide augments adipose macrophage accumulation but is not essential for impaired glucose or insulin tolerance in mice.

http://www.wikidata.org/entity/Q42351778

about

Assessing the human gut microbiota in metabolic diseases Obesity: An Immunometabolic Perspective Immunological contributions to adipose tissue homeostasis LPS-Enhanced Glucose-Stimulated Insulin Secretion Is Normalized by Resveratrol Adipose tissue macrophage in immune regulation of metabolism.Metagenome and metabolism: the tissue microbiota hypothesis.Gut microbioma population: an indicator really sensible to any change in age, diet, metabolic syndrome, and life-style.The intricate association between gut microbiota and development of type 1, type 2 and type 3 diabetes.Microbial translocation and cardiometabolic risk factors in HIV infection.Obesity and microbiota: an example of an intricate relationship.Exploring the influence of the gut microbiota and probiotics on health: a symposium report.Comparison with ancestral diets suggests dense acellular carbohydrates promote an inflammatory microbiota, and may be the primary dietary cause of leptin resistance and obesity Crosstalk between Gut Microbiota and Dietary Lipids Aggravates WAT Inflammation through TLR Signaling.The gut microbiota modulates glycaemic control and serum metabolite profiles in non-obese diabetic mice Modulation of gut microbiota during probiotic-mediated attenuation of metabolic syndrome in high fat diet-fed mice.Childhood obesity: a role for gut microbiota?Intestinal microbiota and faecal transplantation as treatment modality for insulin resistance and type 2 diabetes mellitus.Defective NOD2 peptidoglycan sensing promotes diet-induced inflammation, dysbiosis, and insulin resistance.Understanding the role of gut microbiome in metabolic disease risk.Ganoderma lucidum reduces obesity in mice by modulating the composition of the gut microbiota.Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms.Adaptive Immunity and Antigen-Specific Activation in Obesity-Associated Insulin Resistance.Indigenous microbiota and Leishmaniasis.Influence of gut bacteria on development and progression of non-alcoholic fatty liver disease.Interaction between dietary lipids and gut microbiota regulates hepatic cholesterol metabolism.Investigating a holobiont: Microbiota perturbations and transkingdom networks.The gut microbiota and metabolic disease: current understanding and future perspectives.Diet-microbiota interactions as moderators of human metabolism.The gut microbiota: a key regulator of metabolic diseases.Personalized microbiome-based approaches to metabolic syndrome management and prevention.Diet-induced dysbiosis of the intestinal microbiota and the effects on immunity and disease.Host remodeling of the gut microbiome and metabolic changes during pregnancy Metagenomic Analysis of Antibiotic-Induced Changes in Gut Microbiota in a Pregnant Rat Model Metabolic endotoxemia directly increases the proliferation of adipocyte precursors at the onset of metabolic diseases through a CD14-dependent mechanism Plasma lipopolysaccharide is closely associated with glycemic control and abdominal obesity: evidence from bariatric surgery.Akkermansia muciniphila mediates negative effects of IFNγ on glucose metabolism.Colonic luminal microbiota and bacterial metabolite composition in pregnant Huanjiang mini-pigs: effects of food composition at different times of pregnancy.Potential mediators linking gut bacteria to metabolic health: a critical view.Regulation of metabolism by the innate immune system.Parasites, microbiota and metabolic disease.

P2860

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P2860

Gut-derived lipopolysaccharide augments adipose macrophage accumulation but is not essential for impaired glucose or insulin tolerance in mice.

http://www.wikidata.org/entity/Q42351778

description

2012 nî lūn-bûn

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2012年论文

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2012年论文

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2012年论文

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name

Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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type

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Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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prefLabel

Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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Gut-derived lipopolysaccharide ...... or insulin tolerance in mice.

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Christoph Reinhardt

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Christopher S Reigstad

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Gunnel Östergren Lundén

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Helene Kling Bäckhed

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Maria Ketonen

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Robert Caesar

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P2860

High-fat diet: bacteria interactions promote intestinal inflammation which precedes and correlates with obesity and insulin resistance in mouse

Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance

A core gut microbiome in obese and lean twins

Mechanisms underlying the resistance to diet-induced obesity in germ-free mice

Obesity is associated with macrophage accumulation in adipose tissue

The gut microbiota as an environmental factor that regulates fat storage

Obesity induces a phenotypic switch in adipose tissue macrophage polarization

Metabolic endotoxemia initiates obesity and insulin resistance

Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice

Toll-like receptors and innate immunity

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1701-1707

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10.1136/GUTJNL-2011-301689

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