MMP17/MT4-MMP and thoracic aortic aneurysms: OPNing new potential for effective treatment.

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Particularly interesting Cys-His-rich protein is highly expressed in human intracranial aneurysms and resists aneurysmal rupture.

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MMP17/MT4-MMP and thoracic aortic aneurysms: OPNing new potential for effective treatment.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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MMP17/MT4-MMP and thoracic aor ...... ntial for effective treatment.

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MMP17/MT4-MMP and thoracic aor ...... ntial for effective treatment.

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MMP17/MT4-MMP and thoracic aor ...... ntial for effective treatment.

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MMP17/MT4-MMP and thoracic aor ...... ntial for effective treatment.

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MMP17/MT4-MMP and thoracic aor ...... ntial for effective treatment.

@en

MMP17/MT4-MMP and thoracic aor ...... ntial for effective treatment.

@nl

P1476

MMP17/MT4-MMP and thoracic aortic aneurysms: OPNing new potential for effective treatment

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P2093

Christina L Papke

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Hiromi Yanagisawa

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Losartan, an AT1 antagonist, prevents aortic aneurysm in a mouse model of Marfan syndrome

Remodelling the extracellular matrix in development and disease

Successes and challenges of using whole exome sequencing to identify novel genes underlying an inherited predisposition for thoracic aortic aneurysms and acute aortic dissections

Fibulin-4 deficiency results in ascending aortic aneurysms: a potential link between abnormal smooth muscle cell phenotype and aneurysm progression

Osteopontin is cleaved at multiple sites close to its integrin-binding motifs in milk and is a novel substrate for plasmin and cathepsin D.

Impaired vascular contractility and aortic wall degeneration in fibulin-4 deficient mice: effect of angiotensin II type 1 (AT1) receptor blockade.

Doxycycline delays aneurysm rupture in a mouse model of Marfan syndrome.

MMP-2 regulates Erk1/2 phosphorylation and aortic dilatation in Marfan syndrome

Adhesive properties of osteopontin: regulation by a naturally occurring thrombin-cleavage in close proximity to the GRGDS cell-binding domain.

MFAP5 loss-of-function mutations underscore the involvement of matrix alteration in the pathogenesis of familial thoracic aortic aneurysms and dissections

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109-112

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scholarly article

editorial

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10.1161/CIRCRESAHA.117.306851

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117

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Yoshito Yamashiro

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2015-07-01T00:00:00Z

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26139854

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4493766

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MMP17/MT4-MMP and thoracic aortic aneurysms: OPNing new potential for effective treatment.

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MMP17/MT4-MMP and thoracic aortic aneurysms: OPNing new potential for effective treatment.

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