Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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α2A-adrenergic blockade attenuates septic cardiomyopathy by increasing cardiac norepinephrine concentration and inhibiting cardiac endothelial activation.

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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P1476

Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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P2093

Mu-Ming Yu

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Song-Tao Shou

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Yan-Fen Chai

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Comparison of Dopamine and Norepinephrine in the Treatment of Shock

Nitric oxide and peroxynitrite in health and disease

Sepsis-induced myocardial dysfunction: pathophysiology and management

Circulating Pneumolysin Is a Potent Inducer of Cardiac Injury during Pneumococcal Infection

miR-21-3p controls sepsis-associated cardiac dysfunction via regulating SORBS2.

Nonerythropoietic, tissue-protective peptides derived from the tertiary structure of erythropoietin.

Fluid balance and cardiac function in septic shock as predictors of hospital mortality.

Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy.

Extracellular histones are major mediators of death in sepsis.

Endotoxemia is associated with partial uncoupling of cardiac pacemaker from cholinergic neural control in rats.

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1021

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scholarly article

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10.3389/FIMMU.2017.01021

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Yan-Cun Liu

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2017-08-24T00:00:00Z

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28970829

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Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

about

P2860

P2860

Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments

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type

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P1433

P1476

P2093

P2860

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P2093

P2860

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P577

P698

P932