about
Leptin, a mediator of cardiac damage associated with obesityA role for soluble ST2 in vascular remodeling associated with obesity in rats.The lysyl oxidase inhibitor β-aminopropionitrile reduces body weight gain and improves the metabolic profile in diet-induced obesity in rats.Galectin-3 Blockade Reduces Renal Fibrosis in Two Normotensive Experimental Models of Renal DamageRole for Galectin-3 in Calcific Aortic Valve Stenosis.Beneficial Effects of Galectin-3 Blockade in Vascular and Aortic Valve Alterations in an Experimental Pressure Overload ModelA role for galectin-3 in the development of early molecular alterations in short-term aortic stenosis.Galectin-3 inhibition prevents adipose tissue remodelling in obesity.Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension.Differential proteomics reveals S100-A11 as a key factor in aldosterone-induced collagen expression in human cardiac fibroblasts.Galectin-3 Participates in Cardiovascular Remodeling Associated With Obesity.Neutrophil Gelatinase-Associated Lipocalin, a Novel Mineralocorticoid Biotarget, Mediates Vascular Profibrotic Effects of Mineralocorticoids.The impact of galectin-3 inhibition on aldosterone-induced cardiac and renal injuries.Differential Proteomics Identifies Reticulocalbin-3 as a Novel Negative Mediator of Collagen Production in Human Cardiac Fibroblasts.The endothelial αENaC contributes to vascular endothelial function in vivo.Interleukin-33/ST2 system attenuates aldosterone-induced adipogenesis and inflammation.The role of oxidative stress in the crosstalk between leptin and mineralocorticoid receptor in the cardiac fibrosis associated with obesity.The impact of obesity in the cardiac lipidomic and its consequences in the cardiac damage observed in obese rats.The lysyl oxidase inhibitor (β-aminopropionitrile) reduces leptin profibrotic effects and ameliorates cardiovascular remodeling in diet-induced obesity in rats.Neutrophil Gelatinase-Associated Lipocalin from immune cells is mandatory for aldosterone-induced cardiac remodeling and inflammation.Aldosterone Target NGAL (Neutrophil Gelatinase-Associated Lipocalin) Is Involved in Cardiac Remodeling After Myocardial Infarction Through NFκB Pathway.Inhibition of galectin-3 ameliorates the consequences of cardiac lipotoxicity in a rat model of diet-induced obesity.The impact of bariatric surgery on renal and cardiac functions in morbidly obese patients.Galectin-3 downregulates antioxidant peroxiredoxin-4 in human cardiac fibroblasts: a new pathway to induce cardiac damage?High levels of circulating TNFR1 increase the risk of all-cause mortality and progression of renal disease in type 2 diabetic nephropathy.Aldosterone Impairs Mitochondrial Function in Human Cardiac Fibroblasts via A-Kinase Anchor Protein 12.Leptin induces cardiac fibrosis through galectin-3, mTOR and oxidative stressThe potential role of leptin in the vascular remodeling associated with obesity.Galectin-3 Mediates Aldosterone-Induced Vascular FibrosisRelevance of vascular peroxisome proliferator-activated receptor γ coactivator-1α to molecular alterations in atherosclerosisThe Impact of Cardiac Lipotoxicity on Cardiac Function and Mirnas Signature in Obese and Non-Obese Rats with Myocardial InfarctionA role for fumarate hydratase in mediating oxidative effects of galectin-3 in human cardiac fibroblastsMore than a simple biomarker: the role of NGAL in cardiovascular and renal diseasesThe Crosstalk between Cardiac Lipotoxicity and Mitochondrial Oxidative Stress in the Cardiac Alterations in Diet-Induced Obesity in RatsCT-1 (Cardiotrophin-1)-Gal-3 (Galectin-3) Axis in Cardiac Fibrosis and InflammationMyocardial Injury After Ischemia/Reperfusion Is Attenuated By Pharmacological Galectin-3 InhibitionSoluble ST2 promotes oxidative stress and inflammation in cardiac fibroblasts: an in vitro and in vivo study in aortic stenosisThe role of mitochondrial oxidative stress in the metabolic alterations in diet-induced obesity in rats
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Ernesto Martinez-Martinez
@ast
Ernesto Martinez-Martinez
@en
Ernesto Martinez-Martinez
@es
Ernesto Martinez-Martinez
@nl
Ernesto Martinez-Martinez
@sl
type
label
Ernesto Martinez-Martinez
@ast
Ernesto Martinez-Martinez
@en
Ernesto Martinez-Martinez
@es
Ernesto Martinez-Martinez
@nl
Ernesto Martinez-Martinez
@sl
prefLabel
Ernesto Martinez-Martinez
@ast
Ernesto Martinez-Martinez
@en
Ernesto Martinez-Martinez
@es
Ernesto Martinez-Martinez
@nl
Ernesto Martinez-Martinez
@sl
P1053
M-1376-2017
P106
P21
P31
P3829
P496
0000-0002-3011-2041