S100A8 and S100A9 Are Associated with Doxorubicin-Induced Cardiotoxicity in the Heart of Diabetic Mice.

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S100A8 and S100A9 Are Associated with Doxorubicin-Induced Cardiotoxicity in the Heart of Diabetic Mice.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

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2016年论文

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2016年论文

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2016年论文

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S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

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S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

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S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

@en

S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

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S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

@en

S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

@nl

P1476

S100A8 and S100A9 Are Associat ...... in the Heart of Diabetic Mice.

@en

P2093

Benjamin Y Yung

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Bjorn T Tam

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Feng F Wang

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Thomas K Sin

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Xiao M Pei

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P2860

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S100A8 and S100A9 mediate endotoxin-induced cardiomyocyte dysfunction via the receptor for advanced glycation end products

Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE-/- mice

Congestive heart failure in patients treated with doxorubicin: a retrospective analysis of three trials.

Recommendations for chamber quantification.

S100A8/A9 activate key genes and pathways in colon tumor progression.

S100A8 and S100A9 induce cytokine expression and regulate the NLRP3 inflammasome via ROS-dependent activation of NF-κB(1.).

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334

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scholarly article

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10.3389/FPHYS.2016.00334

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P478

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P50

Michael Ying

Lawrence Chan

Christopher Wai Keung Lai

Parco M. Siu

Sze Chuen Cesar Wong

P577

2016-08-05T00:00:00Z

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27547188

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4974484

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