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Mild neonatal hypoxia exacerbates the effects of vitamin-deficient diet on homocysteine metabolism in ratsVitamin B12-impaired metabolism produces apoptosis and Parkinson phenotype in rats expressing the transcobalamin-oleosin chimera in substantia nigra.Methyl donor deficiency affects fetal programming of gastric ghrelin cell organization and function in the rat.Non-injurious neonatal hypoxia confers resistance to brain senescence in aged male rats.Early methyl donor deficiency alters cAMP signaling pathway and neurosteroidogenesis in the cerebellum of female rat pups.Gestational vitamin B deficiency leads to homocysteine-associated brain apoptosis and alters neurobehavioral development in rats.Vitamin B12 deficiency reduces proliferation and promotes differentiation of neuroblastoma cells and up-regulates PP2A, proNGF, and TACE.Molecular and cellular effects of vitamin B12 in brain, myocardium and liver through its role as co-factor of methionine synthase.Folate and fetal programming: a play in epigenomics?Nutritional models of foetal programming and nutrigenomic and epigenomic dysregulations of fatty acid metabolism in the liver and heart.Gestational methyl donor deficiency alters key proteins involved in neurosteroidogenesis in the olfactory bulbs of newborn female rats and is associated with impaired olfactory performance.Late Maternal Folate Supplementation Rescues from Methyl Donor Deficiency-Associated Brain Defects by Restoring Let-7 and miR-34 Pathways.
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description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
@hy
name
Jean-Luc Daval
@ast
Jean-Luc Daval
@en
Jean-Luc Daval
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Jean-Luc Daval
@nl
Jean-Luc Daval
@sl
type
label
Jean-Luc Daval
@ast
Jean-Luc Daval
@en
Jean-Luc Daval
@es
Jean-Luc Daval
@nl
Jean-Luc Daval
@sl
prefLabel
Jean-Luc Daval
@ast
Jean-Luc Daval
@en
Jean-Luc Daval
@es
Jean-Luc Daval
@nl
Jean-Luc Daval
@sl
P106
P21
P31
P496
0000-0001-5646-0469