Glucocorticoids suppress bone formation via the osteoclast. - Wikidata - wikimedia - TriplyDB

Glucocorticoids suppress bone formation via the osteoclast.

Glucocorticoids suppress bone formation via the osteoclast.

http://www.wikidata.org/entity/Q42404475

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Phosphate regulates osteopontin gene transcription Osteoclasts: what do they do and how do they do it?Nuclear receptors in bone physiology and diseases Management of endocrine disease: Secondary osteoporosis: pathophysiology and management Disorders of bone remodeling The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone c-Src links a RANK/αvβ3 integrin complex to the osteoclast cytoskeleton.NFATc1 in mice represses osteoprotegerin during osteoclastogenesis and dissociates systemic osteopenia from inflammation in cherubism.Management of glucocorticoid-induced osteoporosis: clinical data in relation to disease demographics, bone mineral density and fracture risk.Glucocorticoids, osteocytes, and skeletal fragility: the role of bone vascularity.Dissection of platelet and myeloid cell defects by conditional targeting of the beta3-integrin subunit.NIK stabilization in osteoclasts results in osteoporosis and enhanced inflammatory osteolysis.Mediation of nonerosive arthritis in a mouse model of lupus by interferon-alpha-stimulated monocyte differentiation that is nonpermissive of osteoclastogenesis.Global analysis of the impact of environmental perturbation on cis-regulation of gene expression Cdc42 regulates bone modeling and remodeling in mice by modulating RANKL/M-CSF signaling and osteoclast polarization.A rational use of glucocorticoids in patients with early arthritis has a minimal impact on bone mass.Col3.6-HSD2 transgenic mice: a glucocorticoid loss-of-function model spanning early and late osteoblast differentiation.A medium-chain fatty acid, capric acid, inhibits RANKL-induced osteoclast differentiation via the suppression of NF-κB signaling and blocks cytoskeletal organization and survival in mature osteoclasts.β2-Adrenergic receptor signaling in osteoblasts contributes to the catabolic effect of glucocorticoids on bone.Should bisphosphonates be used for long-term treatment of glucocorticoid-induced osteoporosis?Advances in glucocorticoid-induced osteoporosis Minireview: nuclear receptor regulation of osteoclast and bone remodeling.Inhaled corticosteroids and bone health Diseases affecting bone quality: beyond osteoporosis.Glucocorticoid induced osteoblast apoptosis by increasing E4BP4 expression via up-regulation of Bim.Protection against ovariectomy-induced bone loss by tranilast.Osteoprotegerin prevents glucocorticoid-induced osteocyte apoptosis in mice Voluntary wheel running mitigates the stress-induced bone loss in ovariectomized rats.Calpain-6, a target molecule of glucocorticoids, regulates osteoclastic bone resorption via cytoskeletal organization and microtubule acetylation.Suppression of autophagy in osteocytes does not modify the adverse effects of glucocorticoids on cortical bone The split personality of NKT cells in malignancy, autoimmune and allergic disorders Glucocorticoids act directly on osteoclasts to increase their life span and reduce bone density.Calcitonin and prednisolone display antagonistic actions on bone and have synergistic effects in experimental arthritis.Risk factors for prevalent vertebral fractures in black and white female densitometry patients.Bone density and cortical structure after pediatric renal transplantation.Genetic mouse models for bone studies--strengths and limitations Influence of perioperative dexamethasone on delayed union in mandibular fractures: A clinical and radiological study Microarray gene expression profiling of osteoarthritic bone suggests altered bone remodelling, WNT and transforming growth factor-beta/bone morphogenic protein signalling.Curcumin alleviates glucocorticoid-induced osteoporosis through the regulation of the Wnt signaling pathway.Glucocorticoid effects on changes in bone mineral density and cortical structure in childhood nephrotic syndrome

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P2860

Glucocorticoids suppress bone formation via the osteoclast.

http://www.wikidata.org/entity/Q42404475

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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name

Glucocorticoids suppress bone formation via the osteoclast.

@en

Glucocorticoids suppress bone formation via the osteoclast.

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type

label

Glucocorticoids suppress bone formation via the osteoclast.

@en

Glucocorticoids suppress bone formation via the osteoclast.

@nl

prefLabel

Glucocorticoids suppress bone formation via the osteoclast.

@en

Glucocorticoids suppress bone formation via the osteoclast.

@nl

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Glucocorticoids suppress bone formation via the osteoclast.

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F Patrick Ross

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Haibo Zhao

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Hideki Kitaura

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Hyun-Ju Kim

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Judson A Brewer

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Louis J Muglia

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Sandip Bhattacharyya

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P2860

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Bone resorption by osteoclasts

Origin of osteoclasts: mature monocytes and macrophages are capable of differentiating into osteoclasts under a suitable microenvironment prepared by bone marrow-derived stromal cells.

Mice lacking beta3 integrins are osteosclerotic because of dysfunctional osteoclasts

Severely suppressed bone turnover: a potential complication of alendronate therapy.

Promotion of osteoclast survival and antagonism of bisphosphonate-induced osteoclast apoptosis by glucocorticoids.

Clinical review 151: The role of parathyroid hormone in the pathogenesis of glucocorticoid-induced osteoporosis: a re-examination of the evidence.

Osteoclast-derived activity in the coupling of bone formation to resorption.

Fast glucocorticoid actions on brain: back to the future.

Dynamic changes in the osteoclast cytoskeleton in response to growth factors and cell attachment are controlled by beta3 integrin

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2152-2160

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8

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116

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16878176

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