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Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

http://www.wikidata.org/entity/Q42408122

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Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

http://www.wikidata.org/entity/Q42408122

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@en

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@nl

type

label

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@en

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@nl

prefLabel

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@en

Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@nl

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Journal of Crohn's and Colitis

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Anti-TNF Antibodies and Autophagy: A Hidden Nexus for a Successful Therapeutic Response?

@en

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Christoph Becker

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Stefania Vetrano

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P2860

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Optimal Endpoint of Therapy in IBD: An Update on Factors Determining a Successful Drug Withdrawal

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

IRGM Governs the Core Autophagy Machinery to Conduct Antimicrobial Defense

Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production

A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells

A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1

Crohn's disease and the NOD2 gene: a role for paneth cells.

Impaired autophagy of an intracellular pathogen induced by a Crohn's disease associated ATG16L1 variant.

MTMR3 risk allele enhances innate receptor-induced signaling and cytokines by decreasing autophagy and increasing caspase-1 activation

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237-238

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scholarly article

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10.1093/ECCO-JCC/JJV222

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3

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10

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2015-12-08T00:00:00Z

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286445809

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26645640

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4957480

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