Temporal relationship of peroxynitrite-induced oxidative damage, calpain-mediated cytoskeletal degradation and neurodegeneration after traumatic brain injury.
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The cysteine protease cathepsin B is a key drug target and cysteine protease inhibitors are potential therapeutics for traumatic brain injuryTherapeutic window analysis of the neuroprotective effects of cyclosporine A after traumatic brain injuryCysteine proteases as therapeutic targets: does selectivity matter? A systematic review of calpain and cathepsin inhibitorsCathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug CandidateGLP-1 improves neuropathology after murine cold lesion brain traumaNeuroprotective effects of selective N-type VGCC blockade on stretch-injury-induced calcium dynamics in cortical neuronsTemporal and spatial dynamics of peroxynitrite-induced oxidative damage after spinal cord contusion injury.Influence of PARP-1 polymorphisms in patients after traumatic brain injuryTemporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in mice.The effects of cyclosporin-A on axonal conduction deficits following traumatic brain injury in adult ratsNeuroinflammation resulting from covert brain invasion by common viruses - a potential role in local and global neurodegeneration.Antioxidant therapies in traumatic brain and spinal cord injuryLipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.Molecular hydrogen in drinking water protects against neurodegenerative changes induced by traumatic brain injury.Increase in blood-brain barrier permeability, oxidative stress, and activated microglia in a rat model of blast-induced traumatic brain injury.Axonal pathology in traumatic brain injury.Neuroglobin-overexpression reduces traumatic brain lesion size in mice.Location and level of Etk expression in neurons are associated with varied severity of traumatic brain injurySialoglycosylation of RBC in visceral leishmaniasis leads to enhanced oxidative stress, calpain-induced fragmentation of spectrin and hemolysis.A pharmacological analysis of the neuroprotective efficacy of the brain- and cell-permeable calpain inhibitor MDL-28170 in the mouse controlled cortical impact traumatic brain injury model.In vivo monitoring of neuronal loss in traumatic brain injury: a microdialysis study.Mitochondrial protection after traumatic brain injury by scavenging lipid peroxyl radicals.Pharmacological inhibition of lipid peroxidation attenuates calpain-mediated cytoskeletal degradation after traumatic brain injury.Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.The combination of either tempol or FK506 with delayed hypothermia: implications for traumatically induced microvascular and axonal protectionPost-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in ratsThe salutary effects of DHA dietary supplementation on cognition, neuroplasticity, and membrane homeostasis after brain traumaLipid peroxidation in brain or spinal cord mitochondria after injuryMitigation of NADPH Oxidase 2 Activity as a Strategy to Inhibit Peroxynitrite Formation.Targeting the nNOS/peroxynitrite/calpain system to confer neuroprotection and aid functional recovery in a mouse model of TBI.Relationship of nitric oxide synthase induction to peroxynitrite-mediated oxidative damage during the first week after experimental traumatic brain injuryAdministration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo.Evolution of neuronal and astroglial disruption in the peri-contusional cortex of mice revealed by in vivo two-photon imaging.Propofol ameliorates calpain-induced collapsin response mediator protein-2 proteolysis in traumatic brain injury in ratsBrain injury-induced proteolysis is reduced in a novel calpastatin-overexpressing transgenic mouse.Structural and dynamic study of the tetramerization region of non-erythroid alpha-spectrin: a frayed helix revealed by site-directed spin labeling electron paramagnetic resonance.Glutathione peroxidase activity modulates recovery in the injured immature brain.Spinal cord injury reduces the efficacy of pseudorabies virus labeling of sympathetic preganglionic neurons.Pharmacological evidence for a role of peroxynitrite in the pathophysiology of spinal cord injury.Tempol protection of spinal cord mitochondria from peroxynitrite-induced oxidative damage.
P2860
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P2860
Temporal relationship of peroxynitrite-induced oxidative damage, calpain-mediated cytoskeletal degradation and neurodegeneration after traumatic brain injury.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Temporal relationship of perox ...... after traumatic brain injury.
@en
Temporal relationship of perox ...... after traumatic brain injury.
@nl
type
label
Temporal relationship of perox ...... after traumatic brain injury.
@en
Temporal relationship of perox ...... after traumatic brain injury.
@nl
prefLabel
Temporal relationship of perox ...... after traumatic brain injury.
@en
Temporal relationship of perox ...... after traumatic brain injury.
@nl
P2093
P2860
P1476
Temporal relationship of perox ...... after traumatic brain injury.
@en
P2093
Brian M Thompson
Edward D Hall
P2860
P304
P356
10.1016/J.EXPNEUROL.2007.01.023
P407
P577
2007-02-03T00:00:00Z