Inhaled vasodilator therapy in acute lung injury: first, do NO harm? - Wikidata - wikimedia - TriplyDB

Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

http://www.wikidata.org/entity/Q42416175

about

NO: COPD and beyond.UK guidelines for the use of inhaled nitric oxide therapy in adult ICUs. American-European Consensus Conference on ALI/ARDS.

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

http://www.wikidata.org/entity/Q42416175

description

1995 nî lūn-bûn

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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1995年论文

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1995年论文

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name

Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Inhaled vasodilator therapy in acute lung injury: first, do NO harm?

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Brett SJ

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Evans TW

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P2860

Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination

Quantitation of nitrotyrosine levels in lung sections of patients and animals with acute lung injury

Cytokines. 2. Cytokines and lung inflammation: mechanisms of neutrophil recruitment to the lung

Studies on the pathogenesis of the adult respiratory distress syndrome.

Nitrosyl-hemoglobin formation in the blood of animals exposed to nitric oxide.

Randomized double-blind, multicenter study of prostaglandin E1 in patients with the adult respiratory distress syndrome. Prostaglandin E1 Study Group.

Surfactant and the adult respiratory distress syndrome.

New perspectives on basic mechanisms in lung disease. 2. Neutrophil traffic in the lungs: role of haemodynamics, cell adhesion, and deformability.

Ventilation-perfusion distributions in the adult respiratory distress syndrome.

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