Complement-mediated regulation of the IL-17A axis is a central genetic determinant of the severity of experimental allergic asthma. - Wikidata - wikimedia - TriplyDB

Complement-mediated regulation of the IL-17A axis is a central genetic determinant of the severity of experimental allergic asthma.

Complement-mediated regulation of the IL-17A axis is a central genetic determinant of the severity of experimental allergic asthma.

http://www.wikidata.org/entity/Q42423633

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The roles and functional mechanisms of interleukin-17 family cytokines in mucosal immunity Complement mediators: key regulators of airway tissue remodeling in asthma Th17-mediated inflammation in asthma RAGE: a new frontier in chronic airways disease Phospholipase cε, an effector of ras and rap small GTPases, is required for airway inflammatory response in a mouse model of bronchial asthma.Effect of IL-17 receptor A blockade with brodalumab in inflammatory diseases On the Functional Overlap between Complement and Anti-Microbial Peptides Role of Serum Amyloid A, Granulocyte-Macrophage Colony-Stimulating Factor, and Bone Marrow Granulocyte-Monocyte Precursor Expansion in Segmented Filamentous Bacterium-Mediated Protection from Entamoeba histolytica.Genetic and intervention studies implicating complement C3 as a major target for the treatment of periodontitis.Dectin-2 regulates the effector phase of house dust mite-elicited pulmonary inflammation independently from its role in sensitization.Synthetic di-sulfated iduronic acid attenuates asthmatic response by blocking T-cell recruitment to inflammatory sites Evolving Concepts of Asthma.Pooled genome-wide analysis to identify novel risk loci for pediatric allergic asthma The receptor for the complement C3a anaphylatoxin (C3aR) provides host protection against Listeria monocytogenes-induced apoptosis The complex relationship between inflammation and lung function in severe asthma Th17 cells demonstrate stable cytokine production in a proallergic environment Complement and viral pathogenesis Complement system in lung disease.Complement modulation of T cell immune responses during homeostasis and disease.The transcription factor Etv5 controls TH17 cell development and allergic airway inflammation.IL-17 in severe asthma. Where do we stand?Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.The complement anaphylatoxin C3a receptor (C3aR) contributes to the inflammatory response in dextran sulfate sodium (DSS)-induced colitis in mice.Trif-dependent induction of Th17 immunity by lung dendritic cells The endogenous Th17 response in NO2-promoted allergic airway disease is dispensable for airway hyperresponsiveness and distinct from Th17 adoptive transfer.IL-17 attenuates degradation of ARE-mRNAs by changing the cooperation between AU-binding proteins and microRNA16.Dysregulation of complement system and CD4+ T cell activation pathways implicated in allergic response Inhibiting AKT phosphorylation employing non-cytotoxic anthraquinones ameliorates TH2 mediated allergic airways disease and rhinovirus exacerbation.T(H)17 cells mediate pulmonary collateral priming.Chitinase-like proteins promote IL-17-mediated neutrophilia in a tradeoff between nematode killing and host damage.Linkage specificity and role of properdin in activation of the alternative complement pathway by fungal glycans Update in asthma 2010.Exhaled nitric oxide during infancy as a risk factor for asthma and airway hyperreactivity Collaborative interactions between type 2 innate lymphoid cells and antigen-specific CD4+ Th2 cells exacerbate murine allergic airway diseases with prominent eosinophilia The protective role of TLR6 in a mouse model of asthma is mediated by IL-23 and IL-17A.MyD88-dependent production of IL-17F is modulated by the anaphylatoxin C5a via the Akt signaling pathway.Innate and ozone-induced airway hyperresponsiveness in obese mice: role of TNF-α.Distinct Tlr4-expressing cell compartments control neutrophilic and eosinophilic airway inflammation Regulation and function of innate and adaptive interleukin-17-producing cells Impaired Cell Cycle Regulation in a Natural Equine Model of Asthma.

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P2860

Complement-mediated regulation of the IL-17A axis is a central genetic determinant of the severity of experimental allergic asthma.

http://www.wikidata.org/entity/Q42423633

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2010 nî lūn-bûn

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2010年の論文

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Complement-mediated regulation ...... experimental allergic asthma.

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Complement-mediated regulation ...... experimental allergic asthma.

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Complement-mediated regulation ...... experimental allergic asthma.

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Complement-mediated regulation ...... experimental allergic asthma.

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Complement-mediated regulation ...... experimental allergic asthma.

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Complement-mediated regulation ...... experimental allergic asthma.

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Nature Immunology

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Complement-mediated regulation ...... experimental allergic asthma.

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P2093

Alison L Budelsky

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Alyssa A Sproles

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Ian P Lewkowich

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Jennifer R Clark

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Krista Dienger

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Marsha Wills-Karp

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Stephane Lajoie

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Yusuke Suzuki

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Allergenicity resulting from functional mimicry of a Toll-like receptor complex protein

Allergen uptake, activation, and IL-23 production by pulmonary myeloid DCs drives airway hyperresponsiveness in asthma-susceptible mice

Interleukin-13: central mediator of allergic asthma

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

House dust mite allergen induces asthma via Toll-like receptor 4 triggering of airway structural cells.

A protective role for C5a in the development of allergic asthma associated with altered levels of B7-H1 and B7-DC on plasmacytoid dendritic cells

A regulatory role for the C5a anaphylatoxin in type 2 immunity in asthma.

Deficiency of the murine fifth complement component (C5). A 2-base pair gene deletion in a 5'-exon.

IL-17-induced cytokine release in human bronchial epithelial cells in vitro: role of mitogen-activated protein (MAP) kinases

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928-935

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10.1038/NI.1926

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10

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46036786

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20802484

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