Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice. - Wikidata - wikimedia - TriplyDB

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

http://www.wikidata.org/entity/Q42428975

about

Mannose-binding lectin and the balance between immune protection and complication Lack of the pattern recognition molecule mannose-binding lectin increases susceptibility to influenza A virus infection.A novel L-ficolin/mannose-binding lectin chimeric molecule with enhanced activity against Ebola virus.Intradermal immunization with wall teichoic acid (WTA) elicits and augments an anti-WTA IgG response that protects mice from methicillin-resistant Staphylococcus aureus infection independent of mannose-binding lectin status Murine hyperglycemic vasculopathy and cardiomyopathy: whole-genome gene expression analysis predicts cellular targets and regulatory networks influenced by mannose binding lectin Molecular defects in the mannose binding lectin pathway in dermatological disease: Case report and literature review.Recombinant human mannose-binding lectin dampens human alveolar macrophage inflammatory responses to influenza A virus in vitro.The Local and Systemic Inflammatory Response in a Pig Burn Wound Model With a Pivotal Role for Complement.Recombinant chimeric lectins consisting of mannose-binding lectin and L-ficolin are potent inhibitors of influenza A virus compared with mannose-binding lectin.Mannose-binding lectin binds to amyloid β protein and modulates inflammation.Topical application effect of the isolectin hydrogel (Cramoll 1,4) on second-degree burns: experimental model Parkia pendula seed lectin: potential use to treat cutaneous wounds in healthy and immunocompromised mice.Mannose binding lectin gene deficiency increases susceptibility to traumatic brain injury in mice.Investigating the humoral immune response in chronic venous leg ulcer patients colonised with Pseudomonas aeruginosa Point-of-care microfluidic devices for pathogen detection Structural Study of Binding of α-Mannosides to Mannan-Binding Lectins

P2860

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P2860

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

http://www.wikidata.org/entity/Q42428975

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

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2007年论文

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name

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@en

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@nl

type

label

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@en

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@nl

prefLabel

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@en

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@nl

P1433

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P1433

Journal of Investigative Dermatology

P1476

Burn injury reveals altered phenotype in mannan-binding lectin-deficient mice.

@en

P2093

Jens Chr Jensenius

http://www.w3.org/2001/XMLSchema#string

Kazue Takahashi

http://www.w3.org/2001/XMLSchema#string

Mette Møller-Kristensen

http://www.w3.org/2001/XMLSchema#string

P2860

Mannose-binding lectin-deficient mice are susceptible to infection with Staphylococcus aureus

A second serine protease associated with mannan-binding lectin that activates complement

Structural analysis of monosaccharide recognition by rat liver mannose-binding protein

Mannose-binding lectin-deficient mice display defective apoptotic cell clearance but no autoimmune phenotype

Interaction of mannose-binding protein with associated serine proteases: effects of naturally occurring mutations

Early inflammatory cytokine and acute phase protein response under the stress of thermal injury in rats

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

The induction of matrix metalloproteinase and cytokine expression in synovial fibroblasts stimulated with immune cell microparticles.

Activation of the classical complement pathway by mannose-binding protein in association with a novel C1s-like serine protease

Extrahepatic synthesis of complement proteins in inflammation.

P2888

P304

1524-1531

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scholarly article

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10.1038/SJ.JID.5700748

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6

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127

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Michael R. Hamblin

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2007-03-15T00:00:00Z

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6443142

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17363917

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2936508

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